acetylglucosamine has been researched along with Brain Ischemia in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 7 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Jiang, M; Li, Y; Liu, S; Paschen, W; Sheng, H; Warner, DS; Yang, W; Yu, S; Yu, Z | 1 |
Chi, X; Ding, F; Wei, L; Xu, H; Yu, S | 1 |
Cheng, Q; Chi, X; Ding, F; Wei, L; Xu, H; Yang, Y; Yu, S; Zhou, C | 1 |
Cheng, Q; Ding, F; Li, L; Liu, M; Yang, Y; Yu, S | 1 |
Abe, K; Deguchi, K; Deguchi, S; Ikeda, Y; Kono, S; Liu, N; Liu, W; Omote, Y; Yamashita, T; Yunoki, T | 1 |
Chen, X; Deng, A; Ding, F; Zhou, T | 1 |
Liu, S; Paschen, W; Sheng, H; Yang, W; Yu, Z | 1 |
7 other study(ies) available for acetylglucosamine and Brain Ischemia
Article | Year |
---|---|
XBP1 (X-Box-Binding Protein-1)-Dependent O-GlcNAcylation Is Neuroprotective in Ischemic Stroke in Young Mice and Its Impairment in Aged Mice Is Rescued by Thiamet-G.
Topics: Acetylglucosamine; Age Factors; Animals; Brain Ischemia; Disease Models, Animal; Infarction, Middle Cerebral Artery; Male; Membrane Proteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; Neuroprotection; Protein Folding; Protein Serine-Threonine Kinases; Pyrans; Stroke; Thiazoles; Unfolded Protein Response; X-Box Binding Protein 1 | 2017 |
2-(4-Methoxyphenyl)ethyl-2-acetamido-2-deoxy-β-D-pyranoside, an analog of salidroside, contributes to neuroprotection in cerebral ischemic injury in vitro and in vivo.
Topics: Acetylglucosamine; Animals; Apoptosis; Brain; Brain Ischemia; Capillary Permeability; Cell Hypoxia; Cell Survival; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Glucose; Male; Neurons; Neuroprotective Agents; Primary Cell Culture; Random Allocation; Rats, Sprague-Dawley; Time Factors | 2018 |
2-(4-Methoxyphenyl)ethyl-2-Acetamido-2-deoxy-β-d-pyranoside (A Salidroside Analog) Confers Neuroprotection with a Wide Therapeutic Window by Regulating Local Glucose Metabolism in a Rat Model of Cerebral Ischemic Injury.
Topics: Acetylglucosamine; Animals; Apoptosis; Brain Ischemia; Cerebral Cortex; Corpus Striatum; Disease Models, Animal; Glucose; Male; Neurons; Neuroprotective Agents; Rats, Sprague-Dawley; Stroke | 2018 |
2-(4-Methoxyphenyl)ethyl-2-acetamido-2-deoxy-β-d-pyranoside confers neuroprotection in cell and animal models of ischemic stroke through calpain1/PKA/CREB-mediated induction of neuronal glucose transporter 3.
Topics: Acetylglucosamine; Animals; Brain Ischemia; Calpain; Cyclic AMP Response Element-Binding Protein; Cyclic AMP-Dependent Protein Kinases; Gene Expression Regulation; Glucose Transporter Type 3; Hippocampus; Mice; Molecular Structure; Neurons; Neuroprotective Agents; Rats; Rats, Sprague-Dawley; Stroke | 2014 |
Pericyte protection by edaravone after tissue plasminogen activator treatment in rat cerebral ischemia.
Topics: Acetylglucosamine; Animals; Antipyrine; Astrocytes; Brain Ischemia; Cell Proliferation; Disease Models, Animal; Edaravone; Free Radical Scavengers; Glial Cell Line-Derived Neurotrophic Factor; Glial Fibrillary Acidic Protein; Male; Pericytes; Quinolines; Rats; Rats, Wistar; Receptor, Platelet-Derived Growth Factor beta; Reperfusion; Time Factors; Tissue Plasminogen Activator | 2014 |
Pretreatment with 2-(4-methoxyphenyl)ethyl-2-acetamido-2-deoxy-β-D-pyranoside attenuates cerebral ischemia/reperfusion-induced injury in vitro and in vivo.
Topics: Acetylglucosamine; Animals; Apoptosis; Brain Ischemia; Cell Line; Cell Survival; Male; Mice; Mice, Inbred Strains; Neuroprotective Agents; Nitric Oxide; Nitric Oxide Synthase Type II; Protective Agents; Reperfusion Injury | 2014 |
O-linked β-N-acetylglucosamine modification of proteins is activated in post-ischemic brains of young but not aged mice: Implications for impaired functional recovery from ischemic stress.
Topics: Acetylglucosamine; Aging; Animals; Brain; Brain Ischemia; Male; Mice; Mice, Inbred C57BL; Myocardial Ischemia; Protein Processing, Post-Translational; Psychomotor Performance; Recovery of Function; Stress, Physiological; Stroke; SUMO-1 Protein; Ubiquitination; Unfolded Protein Response | 2016 |