acetylcysteine and Heart Defects, Congenital

acetylcysteine has been researched along with Heart Defects, Congenital in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (16.67)29.6817
2010's2 (33.33)24.3611
2020's3 (50.00)2.80

Authors

AuthorsStudies
Aniagu, S; Chen, T; Huang, Y; Ji, C; Jiang, Y; Tao, Y; Zhang, J1
Chen, J; Chen, T; Jiang, Y; Zhang, M1
Aniagu, S; Chen, T; Ji, C; Jiang, Y; Jin, H; Ren, F; Tong, J1
Feng, Q; Gittenberger-de Groot, AC; Lu, X; Ma, NL; Moazzen, H; Urquhart, BL; Velenosi, TJ; Wisse, LJ1
AlMatar, M; Batool, T; Makky, EA1
Eriksson, UJ; Gittenberger-De Groot, AC; Molin, DG; Poelmann, RE; Roest, PA; Steegers-Theunissen, RP; van Iperen, L; Vis, S; Wisse, LJ1

Reviews

1 review(s) available for acetylcysteine and Heart Defects, Congenital

ArticleYear
Therapeutic Potential of N-Acetylcysteine for Wound Healing, Acute Bronchiolitis, and Congenital Heart Defects.
    Current drug metabolism, 2016, Volume: 17, Issue:2

    Topics: Acetylcysteine; Animals; Antioxidants; Bronchiolitis; Heart Defects, Congenital; Humans; Reactive Oxygen Species; Wound Healing

2016

Other Studies

5 other study(ies) available for acetylcysteine and Heart Defects, Congenital

ArticleYear
AHR/ROS-mediated mitochondria apoptosis contributes to benzo[a]pyrene-induced heart defects and the protective effects of resveratrol.
    Toxicology, 2021, Volume: 462

    Topics: Acetylcysteine; Animals; Apoptosis; Azo Compounds; Benzo(a)pyrene; Dose-Response Relationship, Drug; Heart Defects, Congenital; Membrane Potential, Mitochondrial; Mitochondria; Oxidative Stress; Pyrazoles; Reactive Oxygen Species; Receptors, Aryl Hydrocarbon; Resveratrol; Zebrafish

2021
Fine particulate matter induces heart defects via AHR/ROS-mediated endoplasmic reticulum stress.
    Chemosphere, 2022, Volume: 307, Issue:Pt 2

    Topics: Acetylcysteine; Animals; Apoptosis; Butylamines; Cardiotoxicity; Endoplasmic Reticulum Stress; Heart Defects, Congenital; Hydrocarbons, Aromatic; Particulate Matter; Pharmaceutical Preparations; Reactive Oxygen Species; Zebrafish

2022
AHR-mediated oxidative stress contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish embryos.
    Journal of hazardous materials, 2020, 03-05, Volume: 385

    Topics: Acetylcysteine; Animals; Azo Compounds; Cardiotoxicity; DNA Damage; Embryo, Nonmammalian; Embryonic Development; Heart; Heart Defects, Congenital; Oxidative Stress; Purines; Pyrazoles; Reactive Oxygen Species; Receptors, Aryl Hydrocarbon; Trichloroethylene; Zebrafish; Zebrafish Proteins

2020
N-Acetylcysteine prevents congenital heart defects induced by pregestational diabetes.
    Cardiovascular diabetology, 2014, Feb-18, Volume: 13

    Topics: Acetylcysteine; Animals; Blood Glucose; Cardiotonic Agents; Diabetes Mellitus, Experimental; Female; Heart Defects, Congenital; Male; Mice; Mice, Inbred C57BL; Pregnancy; Pregnancy in Diabetics

2014
Exposure of neural crest cells to elevated glucose leads to congenital heart defects, an effect that can be prevented by N-acetylcysteine.
    Birth defects research. Part A, Clinical and molecular teratology, 2007, Volume: 79, Issue:3

    Topics: Acetylcysteine; Animals; Chick Embryo; Glucose; Heart; Heart Defects, Congenital; Neural Crest

2007