acetylcysteine has been researched along with Heart Defects, Congenital in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (16.67) | 29.6817 |
2010's | 2 (33.33) | 24.3611 |
2020's | 3 (50.00) | 2.80 |
Authors | Studies |
---|---|
Aniagu, S; Chen, T; Huang, Y; Ji, C; Jiang, Y; Tao, Y; Zhang, J | 1 |
Chen, J; Chen, T; Jiang, Y; Zhang, M | 1 |
Aniagu, S; Chen, T; Ji, C; Jiang, Y; Jin, H; Ren, F; Tong, J | 1 |
Feng, Q; Gittenberger-de Groot, AC; Lu, X; Ma, NL; Moazzen, H; Urquhart, BL; Velenosi, TJ; Wisse, LJ | 1 |
AlMatar, M; Batool, T; Makky, EA | 1 |
Eriksson, UJ; Gittenberger-De Groot, AC; Molin, DG; Poelmann, RE; Roest, PA; Steegers-Theunissen, RP; van Iperen, L; Vis, S; Wisse, LJ | 1 |
1 review(s) available for acetylcysteine and Heart Defects, Congenital
Article | Year |
---|---|
Therapeutic Potential of N-Acetylcysteine for Wound Healing, Acute Bronchiolitis, and Congenital Heart Defects.
Topics: Acetylcysteine; Animals; Antioxidants; Bronchiolitis; Heart Defects, Congenital; Humans; Reactive Oxygen Species; Wound Healing | 2016 |
5 other study(ies) available for acetylcysteine and Heart Defects, Congenital
Article | Year |
---|---|
AHR/ROS-mediated mitochondria apoptosis contributes to benzo[a]pyrene-induced heart defects and the protective effects of resveratrol.
Topics: Acetylcysteine; Animals; Apoptosis; Azo Compounds; Benzo(a)pyrene; Dose-Response Relationship, Drug; Heart Defects, Congenital; Membrane Potential, Mitochondrial; Mitochondria; Oxidative Stress; Pyrazoles; Reactive Oxygen Species; Receptors, Aryl Hydrocarbon; Resveratrol; Zebrafish | 2021 |
Fine particulate matter induces heart defects via AHR/ROS-mediated endoplasmic reticulum stress.
Topics: Acetylcysteine; Animals; Apoptosis; Butylamines; Cardiotoxicity; Endoplasmic Reticulum Stress; Heart Defects, Congenital; Hydrocarbons, Aromatic; Particulate Matter; Pharmaceutical Preparations; Reactive Oxygen Species; Zebrafish | 2022 |
AHR-mediated oxidative stress contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish embryos.
Topics: Acetylcysteine; Animals; Azo Compounds; Cardiotoxicity; DNA Damage; Embryo, Nonmammalian; Embryonic Development; Heart; Heart Defects, Congenital; Oxidative Stress; Purines; Pyrazoles; Reactive Oxygen Species; Receptors, Aryl Hydrocarbon; Trichloroethylene; Zebrafish; Zebrafish Proteins | 2020 |
N-Acetylcysteine prevents congenital heart defects induced by pregestational diabetes.
Topics: Acetylcysteine; Animals; Blood Glucose; Cardiotonic Agents; Diabetes Mellitus, Experimental; Female; Heart Defects, Congenital; Male; Mice; Mice, Inbred C57BL; Pregnancy; Pregnancy in Diabetics | 2014 |
Exposure of neural crest cells to elevated glucose leads to congenital heart defects, an effect that can be prevented by N-acetylcysteine.
Topics: Acetylcysteine; Animals; Chick Embryo; Glucose; Heart; Heart Defects, Congenital; Neural Crest | 2007 |