acetylcysteine has been researched along with Diabetic Cardiomyopathies in 12 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 8 (66.67) | 24.3611 |
| 2020's | 4 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Ali, T; Arshad, M; Bashir, Z; Fatima, T; Ishtiaq, A; Khan, U; Khan, W; Murtaza, I; Mushtaq, I; Nawaz, M; Sarwar, M; Tabassum, S; Valadi, H | 1 |
| Chen, R; Leng, Y; Li, W; Xia, Z; Xiong, Y; Xue, R | 1 |
| Cai, L; Chen, XX; Keller, BB; Lin, Q; Tan, Y; Wang, X; Yu, HT; Zheng, Y | 1 |
| Dias, SC; Dludla, PV; Johnson, R; Nkambule, BB | 1 |
| Wang, H | 1 |
| Akman, D; Catalucci, D; Turan, B; Yildirim, SS | 1 |
| Chen, KL; Dong, X; Liu, ZW; Qiu, C; Wei, J; Xue, JH; Zhu, HT | 1 |
| Adams, B; Essop, MF; Mapanga, RF | 1 |
| Duan, YY; Liu, C; Lu, XZ; Ma, J; Shen, MZ; Xing, CY; Yuan, LJ | 1 |
| Bilal, M; Haseeb, A; Khan, MA | 1 |
| Lei, S; Lian, Q; Liu, H; Su, W; Xia, Z; Xia, ZY; Xu, J; Zhan, L; Zhang, Q; Zhang, Y; Zhu, Q | 1 |
| Gao, X; Irwin, MG; Lei, S; Liu, Y; Mao, X; Vanhoutte, PM; Wang, T; Wong, GT; Xia, Z | 1 |
12 other study(ies) available for acetylcysteine and Diabetic Cardiomyopathies
| Article | Year |
|---|---|
N-Acetyl Cysteine, Selenium, and Ascorbic Acid Rescue Diabetic Cardiac Hypertrophy via Mitochondrial-Associated Redox Regulators.
Topics: Acetylcysteine; Animals; Antioxidants; Apoptosis; Apoptosis Regulatory Proteins; Ascorbic Acid; Biomarkers; Blood Glucose; Body Weight; Calcium; Cardiomegaly; Cardiotonic Agents; Cytochromes c; Diabetic Cardiomyopathies; Disease Models, Animal; Down-Regulation; GATA4 Transcription Factor; Lipid Peroxidation; Lipids; Mitochondria, Heart; Myocardium; Oxidation-Reduction; Oxidative Stress; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; PPAR alpha; Rats, Sprague-Dawley; Reactive Oxygen Species; RNA, Messenger; Selenium | 2021 |
Mechanism of N-acetylcysteine in alleviating diabetic myocardial ischemia reperfusion injury by regulating PTEN/Akt pathway through promoting DJ-1.
Topics: Acetylcysteine; Animals; Antioxidants; Apoptosis; Apoptosis Regulatory Proteins; Cell Line; Diabetic Cardiomyopathies; Disease Models, Animal; Male; Myocardial Reperfusion Injury; Myocytes, Cardiac; Oxidative Stress; Protein Deglycase DJ-1; Proto-Oncogene Proteins c-akt; PTEN Phosphohydrolase; Rats; Rats, Sprague-Dawley; Signal Transduction | 2020 |
Engineered cardiac tissues: a novel in vitro model to investigate the pathophysiology of mouse diabetic cardiomyopathy.
Topics: Acetylcysteine; Animals; Benzamides; Cells, Cultured; Diabetic Cardiomyopathies; Glycation End Products, Advanced; Inflammation; Mice; Myocardium; Myocytes, Cardiac; Oxidative Stress; Receptor for Advanced Glycation End Products; Tissue Engineering | 2021 |
Cardioprotective potential of N-acetyl cysteine against hyperglycaemia-induced oxidative damage: a protocol for a systematic review.
Topics: Acetylcysteine; Cardiotonic Agents; Diabetic Cardiomyopathies; Humans; Hyperglycemia; Oxidative Stress; Research Design; Review Literature as Topic; Systematic Reviews as Topic; Treatment Outcome | 2017 |
N-acetyl-L-cysteine relieves myocardial injury in type 2 diabetic rats via inhibiting oxidative stress.
Topics: Acetylcysteine; Animals; Antioxidants; Body Weight; Diabetes Mellitus, Experimental; Diabetes Mellitus, Type 2; Diabetic Cardiomyopathies; Humans; Oxidative Stress; Random Allocation; Rats; Rats, Inbred Strains | 2020 |
Relationship between downregulation of miRNAs and increase of oxidative stress in the development of diabetic cardiac dysfunction: junctin as a target protein of miR-1.
Topics: Acetylcysteine; Animals; Carrier Proteins; Cells, Cultured; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Down-Regulation; Gene Expression Regulation; Male; Mice; MicroRNAs; Muscle Proteins; Myocardium; Myocytes, Cardiac; Nitric Oxide; Oxidative Stress; Rats; Rats, Wistar | 2013 |
Protein kinase RNA-like endoplasmic reticulum kinase (PERK) signaling pathway plays a major role in reactive oxygen species (ROS)-mediated endoplasmic reticulum stress-induced apoptosis in diabetic cardiomyopathy.
Topics: Acetylcysteine; Activating Transcription Factor 6; Animals; Apoptosis; Cells, Cultured; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Disease Models, Animal; eIF-2 Kinase; Endoplasmic Reticulum Stress; Free Radical Scavengers; Gene Knockdown Techniques; Glucose; Membrane Proteins; Myocytes, Cardiac; Protein Serine-Threonine Kinases; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Signal Transduction | 2013 |
Partial inhibition of the ubiquitin-proteasome system ameliorates cardiac dysfunction following ischemia-reperfusion in the presence of high glucose.
Topics: Acetylcysteine; Animals; Anti-Inflammatory Agents; Antioxidants; Autophagy; Diabetic Cardiomyopathies; Hyperglycemia; Inflammation Mediators; Isolated Heart Preparation; Leupeptins; Male; Myocardial Reperfusion Injury; Myocardium; Proteasome Inhibitors; Rats, Wistar; Superoxide Dismutase; Superoxide Dismutase-1; Time Factors | 2015 |
N-Acetyl Cysteine improves the diabetic cardiac function: possible role of fibrosis inhibition.
Topics: Acetylcysteine; Animals; Cells, Cultured; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Fibrosis; Male; Mice; Mice, Inbred C57BL; Oxidative Stress | 2015 |
N-Acetyl Cysteine: A Possible Treatment for Diabetic Cardiomyopathy.
Topics: Acetylcysteine; Animals; Antiviral Agents; Diabetic Cardiomyopathies; Fibrosis; Mice; Oxidative Stress | 2016 |
N-acetylcysteine attenuates myocardial dysfunction and postischemic injury by restoring caveolin-3/eNOS signaling in diabetic rats.
Topics: Acetylcysteine; Animals; Antioxidants; Cardiomegaly; Caveolae; Caveolin 3; Cell Hypoxia; Cell Line; Cytoprotection; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Heart Rate; Male; Myocardial Reperfusion Injury; Myocytes, Cardiac; Nitric Oxide; Nitric Oxide Synthase Type III; Oxidative Stress; Phosphorylation; Rats, Sprague-Dawley; RNA Interference; Signal Transduction; Streptozocin; Transfection; Ventricular Function, Left | 2016 |
PKCĪ² inhibition with ruboxistaurin reduces oxidative stress and attenuates left ventricular hypertrophy and dysfunction in rats with streptozotocin-induced diabetes.
Topics: Acetylcysteine; Animals; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Dinoprost; Drug Evaluation, Preclinical; Enzyme Activation; Free Radical Scavengers; Hypertrophy, Left Ventricular; Indoles; Isoprostanes; Male; Maleimides; Myocytes, Cardiac; NADPH Oxidases; Oxidative Stress; Protein Kinase C; Protein Kinase C beta; Rats; Rats, Sprague-Dawley; Superoxides; Ultrasonography | 2012 |