acetylcysteine and Carcinoma, Medullary

acetylcysteine has been researched along with Carcinoma, Medullary in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Adler, G; Böhm, BO; Dralle, H; Hoang-Vu, C; Kessler, H; Ludwig, L; Schmid, RM; Wagner, M	1

Other Studies

1 other study(ies) available for acetylcysteine and Carcinoma, Medullary

Article	Year
Nuclear factor-kappaB is constitutively active in C-cell carcinoma and required for RET-induced transformation. Cancer research, 2001, Jun-01, Volume: 61, Issue:11 Topics: 3T3 Cells; Acetylcysteine; Animals; Apoptosis; Carcinoma, Medullary; Cell Nucleus; Cell Transformation, Neoplastic; Dimerization; DNA-Binding Proteins; DNA, Neoplasm; Drosophila Proteins; Fibroblasts; Humans; I-kappa B Kinase; I-kappa B Proteins; MAP Kinase Signaling System; Mice; Mitogen-Activated Protein Kinases; NF-kappa B; NF-KappaB Inhibitor alpha; Protein Serine-Threonine Kinases; Proto-Oncogene Mas; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-ret; ras Proteins; Receptor Protein-Tyrosine Kinases; Signal Transduction; Thyroid Neoplasms; Transcription Factor RelA; Transcription, Genetic; Tumor Cells, Cultured	2001

Article

Year

Nuclear factor-kappaB is constitutively active in C-cell carcinoma and required for RET-induced transformation.

Cancer research, 2001, Jun-01, Volume: 61, Issue:11

Topics: 3T3 Cells; Acetylcysteine; Animals; Apoptosis; Carcinoma, Medullary; Cell Nucleus; Cell Transformation, Neoplastic; Dimerization; DNA-Binding Proteins; DNA, Neoplasm; Drosophila Proteins; Fibroblasts; Humans; I-kappa B Kinase; I-kappa B Proteins; MAP Kinase Signaling System; Mice; Mitogen-Activated Protein Kinases; NF-kappa B; NF-KappaB Inhibitor alpha; Protein Serine-Threonine Kinases; Proto-Oncogene Mas; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-ret; ras Proteins; Receptor Protein-Tyrosine Kinases; Signal Transduction; Thyroid Neoplasms; Transcription Factor RelA; Transcription, Genetic; Tumor Cells, Cultured

2001