Page last updated: 2024-08-17

acetyl coenzyme a and Recrudescence

acetyl coenzyme a has been researched along with Recrudescence in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Brindle, PK; Chadburn, A; Chiarenza, A; Dalla-Favera, R; Dominguez-Sola, D; Fabbri, G; Gaidano, G; Grunn, A; Kasper, LH; Lerach, S; Ma, J; Mullighan, CG; Murty, VV; Pasqualucci, L; Rabadan, R; Rossi, D; Tang, H; Trifonov, V	1
Ertel, A; Flomenberg, N; Howell, A; Knudsen, ES; Lin, Z; Lisanti, MP; Martinez-Outschoorn, UE; Pavlides, S; Pestell, RG; Prisco, M; Sotgia, F; Tsirigos, A; Wang, C	1

Other Studies

2 other study(ies) available for acetyl coenzyme a and Recrudescence

Article	Year
Inactivating mutations of acetyltransferase genes in B-cell lymphoma. Nature, 2011, Mar-10, Volume: 471, Issue:7337 Topics: Acetyl Coenzyme A; Acetylation; Acetyltransferases; Animals; Base Sequence; Cells, Cultured; CREB-Binding Protein; DNA-Binding Proteins; E1A-Associated p300 Protein; Gene Expression Regulation, Neoplastic; HEK293 Cells; Histone Acetyltransferases; Humans; Lymphoma, B-Cell; Lymphoma, Follicular; Lymphoma, Large B-Cell, Diffuse; Mice; Mutation; Mutation, Missense; Polymorphism, Single Nucleotide; Protein Binding; Protein Structure, Tertiary; Proto-Oncogene Proteins c-bcl-6; Recurrence; Sequence Deletion; Tumor Suppressor Protein p53	2011
Ketones and lactate increase cancer cell "stemness," driving recurrence, metastasis and poor clinical outcome in breast cancer: achieving personalized medicine via Metabolo-Genomics. Cell cycle (Georgetown, Tex.), 2011, Apr-15, Volume: 10, Issue:8 Topics: Acetyl Coenzyme A; Biomarkers, Tumor; Breast Neoplasms; Cell Line, Tumor; Citric Acid Cycle; Female; Fibroblasts; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Genome-Wide Association Study; Genomics; Glycolysis; Humans; Ketones; Lactic Acid; Metabolomics; Mitochondria; Neoplasm Metastasis; Oxidative Phosphorylation; Precision Medicine; Recurrence; Stem Cells; Treatment Outcome	2011

Article

Year

Inactivating mutations of acetyltransferase genes in B-cell lymphoma.

Nature, 2011, Mar-10, Volume: 471, Issue:7337

Topics: Acetyl Coenzyme A; Acetylation; Acetyltransferases; Animals; Base Sequence; Cells, Cultured; CREB-Binding Protein; DNA-Binding Proteins; E1A-Associated p300 Protein; Gene Expression Regulation, Neoplastic; HEK293 Cells; Histone Acetyltransferases; Humans; Lymphoma, B-Cell; Lymphoma, Follicular; Lymphoma, Large B-Cell, Diffuse; Mice; Mutation; Mutation, Missense; Polymorphism, Single Nucleotide; Protein Binding; Protein Structure, Tertiary; Proto-Oncogene Proteins c-bcl-6; Recurrence; Sequence Deletion; Tumor Suppressor Protein p53

2011

Ketones and lactate increase cancer cell "stemness," driving recurrence, metastasis and poor clinical outcome in breast cancer: achieving personalized medicine via Metabolo-Genomics.

Cell cycle (Georgetown, Tex.), 2011, Apr-15, Volume: 10, Issue:8

Topics: Acetyl Coenzyme A; Biomarkers, Tumor; Breast Neoplasms; Cell Line, Tumor; Citric Acid Cycle; Female; Fibroblasts; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Genome-Wide Association Study; Genomics; Glycolysis; Humans; Ketones; Lactic Acid; Metabolomics; Mitochondria; Neoplasm Metastasis; Oxidative Phosphorylation; Precision Medicine; Recurrence; Stem Cells; Treatment Outcome

2011