acetyl coenzyme a has been researched along with Pancreatic Neoplasms in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Bai, J; Chen, J; Gao, C; Li, H; Meng, Z; Shen, Q; Wu, H; Wu, S; Yin, T; Zhang, H | 1 |
Affronti, HC; Campbell, SL; Carrer, A; Egolf, S; Garcia, BA; Gardini, A; Norgard, RJ; Parris, JLD; Schultz, KC; Sela, Y; Sidoli, S; Sivanand, S; Snyder, NW; Stanger, BZ; Trefely, S; Trizzino, M; Wellen, KE; Zhao, S | 1 |
Halbrook, CJ; Lysstiotis, CA; Nelson, BS | 1 |
Ahmed, S; Bassilian, S; Boren, J; Boros, LG; Cascante, M; Centelles, JJ; Lee, WN; Lim, S | 1 |
4 other study(ies) available for acetyl coenzyme a and Pancreatic Neoplasms
Article | Year |
---|---|
Hyperglycemia Enhances Immunosuppression and Aerobic Glycolysis of Pancreatic Cancer Through Upregulating Bmi1-UPF1-HK2 Pathway.
Topics: Acetyl Coenzyme A; Animals; Deoxyglucose; Glucose; Glycolysis; Histones; Humans; Hyperglycemia; Immunosuppression Therapy; Mice; Mice, Nude; Pancreatic Neoplasms; Polycomb Repressive Complex 1; RNA, Messenger; Streptozocin; Tumor Microenvironment | 2022 |
Acetyl-CoA Metabolism Supports Multistep Pancreatic Tumorigenesis.
Topics: Acetyl Coenzyme A; Acetylation; Acinar Cells; Animals; Carcinogenesis; Carcinoma, Pancreatic Ductal; Cell Proliferation; Female; Genes, ras; Heterografts; Histones; Humans; Male; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Mutation; Pancreatic Neoplasms; Protein Processing, Post-Translational; Signal Transduction | 2019 |
Metabolism Drives Carcinogenesis and Maintenance of Pancreatic Tumors.
Topics: Acetyl Coenzyme A; Carcinogenesis; Drive; Humans; Maintenance; Pancreatic Neoplasms | 2019 |
The stable isotope-based dynamic metabolic profile of butyrate-induced HT29 cell differentiation.
Topics: Acetyl Coenzyme A; Adenocarcinoma; Butyrates; Carbon Isotopes; Cell Differentiation; Cell Division; Colonic Neoplasms; Glucose; Glycolysis; HT29 Cells; Humans; Malonyl Coenzyme A; Oxidation-Reduction; Pancreatic Neoplasms; Pentose Phosphate Pathway | 2003 |