Page last updated: 2024-08-17

acetyl coenzyme a and Lymphoma, Large B-Cell, Diffuse

acetyl coenzyme a has been researched along with Lymphoma, Large B-Cell, Diffuse in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cantley, LC; Cerchietti, L; Chen, Z; Chiang, YL; Cross, JR; Duy, C; Green, MR; Hong, JY; Hu, J; Jain, N; Jing, H; Li, M; Lin, H; Lyssiotis, CA; Melnick, AM; Mistry, SJ; Shen, H; Teater, MR; Wang, L1
Brindle, PK; Chadburn, A; Chiarenza, A; Dalla-Favera, R; Dominguez-Sola, D; Fabbri, G; Gaidano, G; Grunn, A; Kasper, LH; Lerach, S; Ma, J; Mullighan, CG; Murty, VV; Pasqualucci, L; Rabadan, R; Rossi, D; Tang, H; Trifonov, V1

Other Studies

2 other study(ies) available for acetyl coenzyme a and Lymphoma, Large B-Cell, Diffuse

ArticleYear
Non-oncogene Addiction to SIRT3 Plays a Critical Role in Lymphomagenesis.
    Cancer cell, 2019, 06-10, Volume: 35, Issue:6

    Topics: Acetyl Coenzyme A; Animals; Antineoplastic Agents; Autophagic Cell Death; Cell Proliferation; Citric Acid Cycle; Energy Metabolism; Female; Gene Expression Regulation, Enzymologic; Gene Expression Regulation, Neoplastic; Glutamine; HEK293 Cells; Histone Deacetylase Inhibitors; Humans; Lymphoma, Large B-Cell, Diffuse; MCF-7 Cells; Mice, Inbred C57BL; Mice, Inbred NOD; Mice, Knockout; Mice, SCID; Molecular Targeted Therapy; Signal Transduction; Sirtuin 3; Xenograft Model Antitumor Assays

2019
Inactivating mutations of acetyltransferase genes in B-cell lymphoma.
    Nature, 2011, Mar-10, Volume: 471, Issue:7337

    Topics: Acetyl Coenzyme A; Acetylation; Acetyltransferases; Animals; Base Sequence; Cells, Cultured; CREB-Binding Protein; DNA-Binding Proteins; E1A-Associated p300 Protein; Gene Expression Regulation, Neoplastic; HEK293 Cells; Histone Acetyltransferases; Humans; Lymphoma, B-Cell; Lymphoma, Follicular; Lymphoma, Large B-Cell, Diffuse; Mice; Mutation; Mutation, Missense; Polymorphism, Single Nucleotide; Protein Binding; Protein Structure, Tertiary; Proto-Oncogene Proteins c-bcl-6; Recurrence; Sequence Deletion; Tumor Suppressor Protein p53

2011