acetovanillone has been researched along with Blood Poisoning in 6 studies
apocynin : An aromatic ketone that is 1-phenylethanone substituted by a hydroxy group at position 4 and a methoxy group at position 3.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Combination therapy with niacin and apocynin significantly improved survival in rats with sepsis (75." | 8.31 | Combination Therapy of Niacin and Apocynin Attenuates Lung Injury During Sepsis in Rats. ( Jung, YS; Kim, H; Kim, KS; Kim, T; Kwon, WY; Park, H; Shin, J; Suh, GJ, 2023) |
| "Combination therapy with niacin and apocynin significantly improved survival in rats with sepsis (75." | 4.31 | Combination Therapy of Niacin and Apocynin Attenuates Lung Injury During Sepsis in Rats. ( Jung, YS; Kim, H; Kim, KS; Kim, T; Kwon, WY; Park, H; Shin, J; Suh, GJ, 2023) |
| "Sepsis resulted in cognitive impairments, which was accompanied by selective phenotype loss of PV interneurons and increased gp91(phox), 4-hydroxynonenal, malondialdehyde, IL-1β, and IL-6 expressions." | 3.81 | Sepsis-induced selective parvalbumin interneuron phenotype loss and cognitive impairments may be mediated by NADPH oxidase 2 activation in mice. ( Ji, MH; Jia, M; Ju, LS; Qiu, LL; Shen, JC; Sun, XR; Tang, H; Yang, JJ; Zhang, H; Zuo, ZY, 2015) |
| "To determine the roles of nitric oxide synthase (NOS) and nicotinamide adenine dinucleotide phosphate (NADPH) oxidase in the impairment of capillary blood flow in sepsis and in the reversal of this impairment by ascorbate." | 3.74 | Septic impairment of capillary blood flow requires nicotinamide adenine dinucleotide phosphate oxidase but not nitric oxide synthase and is rapidly reversed by ascorbate through an endothelial nitric oxide synthase-dependent mechanism. ( Li, F; Tyml, K; Wilson, JX, 2008) |
| "Sepsis was induced by cecal ligation and puncture (CLP)." | 1.56 | Perivascular adipose tissue phenotype and sepsis vascular dysfunction: Differential contribution of NO, ROS and beta 3-adrenergic receptor. ( Assreuy, J; Barp, CG; Benedet, PO, 2020) |
| "Sepsis was induced in WT and gp91(phox) knockout mice (gp91(phox-/-)) by cecal ligation and puncture (CLP) to induce fecal peritonitis." | 1.40 | The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. ( Bozza, FA; Britto, LR; Castro-Faria-Neto, HC; Cunha, FQ; D'Avila, JC; Hernandes, MS; Kinjo, ER; Lopes, LR; Reis, PA; Trevelin, SC, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (16.67) | 29.6817 |
| 2010's | 3 (50.00) | 24.3611 |
| 2020's | 2 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Park, H | 1 |
| Jung, YS | 1 |
| Suh, GJ | 1 |
| Kwon, WY | 1 |
| Kim, KS | 1 |
| Kim, T | 1 |
| Kim, H | 1 |
| Shin, J | 1 |
| Barp, CG | 1 |
| Benedet, PO | 1 |
| Assreuy, J | 1 |
| Hernandes, MS | 1 |
| D'Avila, JC | 1 |
| Trevelin, SC | 2 |
| Reis, PA | 1 |
| Kinjo, ER | 1 |
| Lopes, LR | 2 |
| Castro-Faria-Neto, HC | 1 |
| Cunha, FQ | 2 |
| Britto, LR | 1 |
| Bozza, FA | 1 |
| Ji, MH | 1 |
| Qiu, LL | 1 |
| Tang, H | 1 |
| Ju, LS | 1 |
| Sun, XR | 1 |
| Zhang, H | 1 |
| Jia, M | 1 |
| Zuo, ZY | 1 |
| Shen, JC | 1 |
| Yang, JJ | 1 |
| Dos Santos, CX | 1 |
| Ferreira, RG | 1 |
| de Sá Lima, L | 1 |
| Silva, RL | 1 |
| Scavone, C | 1 |
| Curi, R | 1 |
| Alves-Filho, JC | 1 |
| Cunha, TM | 1 |
| Roxo-Júnior, P | 1 |
| Cervi, MC | 1 |
| Laurindo, FR | 1 |
| Hothersall, JS | 1 |
| Cobb, AM | 1 |
| Zhang, M | 1 |
| Ivetic, A | 1 |
| Shah, AM | 1 |
| Tyml, K | 1 |
| Li, F | 1 |
| Wilson, JX | 1 |
6 other studies available for acetovanillone and Blood Poisoning
| Article | Year |
|---|---|
Combination Therapy of Niacin and Apocynin Attenuates Lung Injury During Sepsis in Rats.
Topics: Acetophenones; Animals; Glutathione; Lung; Lung Injury; Male; NADP; NADPH Oxidases; NF-kappa B; Niac | 2023 |
Perivascular adipose tissue phenotype and sepsis vascular dysfunction: Differential contribution of NO, ROS and beta 3-adrenergic receptor.
Topics: Acetophenones; Adipose Tissue; Adrenergic beta-3 Receptor Agonists; Animals; Aorta; Cyclic N-Oxides; | 2020 |
The role of Nox2-derived ROS in the development of cognitive impairment after sepsis.
Topics: Acetophenones; Animals; Anti-Inflammatory Agents, Non-Steroidal; Avoidance Learning; Bacterial Prote | 2014 |
Sepsis-induced selective parvalbumin interneuron phenotype loss and cognitive impairments may be mediated by NADPH oxidase 2 activation in mice.
Topics: Acetophenones; Animals; Anti-Inflammatory Agents, Non-Steroidal; Cells, Cultured; Cognition Disorder | 2015 |
Apocynin and Nox2 regulate NF-κB by modifying thioredoxin-1 redox-state.
Topics: Acetophenones; Animals; Granulomatous Disease, Chronic; Lipopolysaccharides; Male; Mice; Mice, Knock | 2016 |
Septic impairment of capillary blood flow requires nicotinamide adenine dinucleotide phosphate oxidase but not nitric oxide synthase and is rapidly reversed by ascorbate through an endothelial nitric oxide synthase-dependent mechanism.
Topics: Acetophenones; Animals; Antioxidants; Ascorbic Acid; Capillaries; Male; Mice; Mice, Knockout; Microc | 2008 |