acetaldehyde has been researched along with Inflammation in 32 studies
Acetaldehyde: A colorless, flammable liquid used in the manufacture of acetic acid, perfumes, and flavors. It is also an intermediate in the metabolism of alcohol. It has a general narcotic action and also causes irritation of mucous membranes. Large doses may cause death from respiratory paralysis.
acetaldehyde : The aldehyde formed from acetic acid by reduction of the carboxy group. It is the most abundant carcinogen in tobacco smoke.
aldehyde : A compound RC(=O)H, in which a carbonyl group is bonded to one hydrogen atom and to one R group.
acetyl group : A group, formally derived from acetic acid by dehydroxylation, which is fundamental to the biochemistry of all forms of life. When bound to coenzyme A, it is central to the metabolism of carbohydrates and fats.
Inflammation: A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To determine the effects of a low concentration of acetaldehyde, which itself did not trigger airway inflammation, on extant allergic airway inflammation in a murine model of allergic asthma." | 7.78 | Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound. ( Fukahori, S; Fukushima, C; Kawano, T; Kohno, S; Matsuse, H; Nishino, T; Tsuchida, T, 2012) |
| "Exposure to acetaldehyde can enhance allergic airway inflammation in asthma." | 7.78 | Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound. ( Fukahori, S; Fukushima, C; Kawano, T; Kohno, S; Matsuse, H; Nishino, T; Tsuchida, T, 2012) |
| "The purpose of the present study was to determine the effects of acetaldehyde on airway smooth muscle constriction and inflammation." | 7.74 | Effects of acetaldehyde on human airway constriction and inflammation. ( Fukushima, C; Kohno, S; Matsuse, H; Sadahiro, A; Shimoda, T, 2007) |
| " An oral ethanol provocation test was performed in Japanese asthmatics to measure pulmonary function, blood ethanol, acetaldehyde and histamine." | 7.74 | Effects of acetaldehyde on human airway constriction and inflammation. ( Fukushima, C; Kohno, S; Matsuse, H; Sadahiro, A; Shimoda, T, 2007) |
| "Glycolaldehyde (GA) has been shown to impair cellular function in various disorders, including diabetes, and renal diseases." | 5.72 | Glycolaldehyde induces synergistic effects on vascular inflammation in TNF-α-stimulated vascular smooth muscle cells. ( Choi, IW; Gu, MJ; Ha, SK; Kim, Y; Lee, HW; Lee, SH; Yoo, G, 2022) |
| "Acetaldehyde is an endocrine-disrupting chemical (EDC) and a volatile organic compound (VOC)." | 5.38 | Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound. ( Fukahori, S; Fukushima, C; Kawano, T; Kohno, S; Matsuse, H; Nishino, T; Tsuchida, T, 2012) |
| "Glycolaldehyde was enzymically formed from glyceraldehyde, the fructokinase/aldolase B product of fructose." | 5.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| "Glyoxal was also the Fenton product responsible for glycolaldehyde protein carbonylation as carbonylation was prevented by aminoguanidine or Fenton inhibitors." | 5.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| " Thus fructose was seventy-fold more toxic if hepatocytes were exposed to non-toxic levels of hydrogen peroxide (H(2)O(2)) released by inflammatory cells." | 5.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| "Malondialdehyde-acetaldehyde adducts (MAA) act as potent immune adjuvants and co-localize with citrullinated antigens in tissues effected by rheumatoid arthritis (RA)." | 3.96 | Immunogenic and inflammatory responses to citrullinated proteins are enhanced following modification with malondialdehyde-acetaldehyde adducts. ( Daubach, EC; Duryee, MJ; England, BR; Fletcher, BS; Hunter, CD; Klassen, LW; Mikuls, TR; Pospisil, TP; Thiele, GM, 2020) |
| " We hypothesised that acrolein, nicotine and acetylaldehyde, important chemicals contained within volatile cigarette smoke in terms of inducing inflammation and causing addiction, have immunomodulatory effects in primary nasal epithelial cell cultures (PNECs)." | 3.80 | Inflammatory and cytotoxic effects of acrolein, nicotine, acetylaldehyde and cigarette smoke extract on human nasal epithelial cells. ( Comer, DM; Elborn, JS; Ennis, M, 2014) |
| "To determine the effects of a low concentration of acetaldehyde, which itself did not trigger airway inflammation, on extant allergic airway inflammation in a murine model of allergic asthma." | 3.78 | Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound. ( Fukahori, S; Fukushima, C; Kawano, T; Kohno, S; Matsuse, H; Nishino, T; Tsuchida, T, 2012) |
| "Exposure to acetaldehyde can enhance allergic airway inflammation in asthma." | 3.78 | Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound. ( Fukahori, S; Fukushima, C; Kawano, T; Kohno, S; Matsuse, H; Nishino, T; Tsuchida, T, 2012) |
| " Direct administration of acetate increased nociceptive behaviors suggesting that acetate, not acetaldehyde, accumulation results in hangover-like hypersensitivity in our model." | 3.76 | Acetate causes alcohol hangover headache in rats. ( Hoek, JB; Maxwell, CR; Oshinsky, ML; Silberstein, SD; Spangenberg, RJ, 2010) |
| "The purpose of the present study was to determine the effects of acetaldehyde on airway smooth muscle constriction and inflammation." | 3.74 | Effects of acetaldehyde on human airway constriction and inflammation. ( Fukushima, C; Kohno, S; Matsuse, H; Sadahiro, A; Shimoda, T, 2007) |
| " An oral ethanol provocation test was performed in Japanese asthmatics to measure pulmonary function, blood ethanol, acetaldehyde and histamine." | 3.74 | Effects of acetaldehyde on human airway constriction and inflammation. ( Fukushima, C; Kohno, S; Matsuse, H; Sadahiro, A; Shimoda, T, 2007) |
| " We have recently proposed another mechanism for aldehyde generation at sites of inflammation that involves myeloperoxidase, a heme enzyme secreted by activated phagocytes." | 3.69 | Human neutrophils employ the myeloperoxidase-hydrogen peroxide-chloride system to convert hydroxy-amino acids into glycolaldehyde, 2-hydroxypropanal, and acrolein. A mechanism for the generation of highly reactive alpha-hydroxy and alpha,beta-unsaturated ( Anderson, MM; Hazen, SL; Heinecke, JW; Hsu, FF, 1997) |
| "Inflammation is widely accepted to play a major role in atherosclerosis and other cardiovascular diseases." | 2.52 | Aldehyde-modified proteins as mediators of early inflammation in atherosclerotic disease. ( Anderson, DR; Antoniak, DT; Duryee, MJ; Mikuls, TR; Thiele, GM, 2015) |
| "Glycolaldehyde (GA) has been shown to impair cellular function in various disorders, including diabetes, and renal diseases." | 1.72 | Glycolaldehyde induces synergistic effects on vascular inflammation in TNF-α-stimulated vascular smooth muscle cells. ( Choi, IW; Gu, MJ; Ha, SK; Kim, Y; Lee, HW; Lee, SH; Yoo, G, 2022) |
| "Inflammation is known to induce oxidative stress, which can lead to lipid peroxidation." | 1.62 | Malondialdehyde-Acetaldehyde Modified (MAA) Proteins Differentially Effect the Inflammatory Response in Macrophage, Endothelial Cells and Animal Models of Cardiovascular Disease. ( Anderson, DR; Clemens, DL; Duryee, LM; Duryee, MJ; Garvin, RP; Opperman, PJ; Thiele, GM, 2021) |
| "Acrolein was pro-inflammatory for the PNEC cultures (30 μM exposure for 4 h inducing a 2." | 1.40 | Inflammatory and cytotoxic effects of acrolein, nicotine, acetylaldehyde and cigarette smoke extract on human nasal epithelial cells. ( Comer, DM; Elborn, JS; Ennis, M, 2014) |
| "Acetaldehyde is an endocrine-disrupting chemical (EDC) and a volatile organic compound (VOC)." | 1.38 | Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound. ( Fukahori, S; Fukushima, C; Kawano, T; Kohno, S; Matsuse, H; Nishino, T; Tsuchida, T, 2012) |
| "The mechanism of veisalgia cephalgia or hangover headache is unknown." | 1.36 | Acetate causes alcohol hangover headache in rats. ( Hoek, JB; Maxwell, CR; Oshinsky, ML; Silberstein, SD; Spangenberg, RJ, 2010) |
| "Glycolaldehyde was enzymically formed from glyceraldehyde, the fructokinase/aldolase B product of fructose." | 1.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| "Glyoxal was also the Fenton product responsible for glycolaldehyde protein carbonylation as carbonylation was prevented by aminoguanidine or Fenton inhibitors." | 1.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| " Thus fructose was seventy-fold more toxic if hepatocytes were exposed to non-toxic levels of hydrogen peroxide (H(2)O(2)) released by inflammatory cells." | 1.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| "pHA-lysine was detected in inflamed human tissues that were reduced, hydrolyzed, and then analyzed by mass spectrometry, indicating that the reaction of pHA with proteins may be of physiological importance." | 1.30 | p-Hydroxyphenylacetaldehyde, the major product of L-tyrosine oxidation by the myeloperoxidase-H2O2-chloride system of phagocytes, covalently modifies epsilon-amino groups of protein lysine residues. ( Crowley, JR; d'Avignon, A; Gaut, JP; Hazen, SL; Heinecke, JW; Hsu, FF, 1997) |
| "No acetaldehyde adduct was detected in the control rats that were pair-fed with isocaloric amounts of dextrose." | 1.30 | Acetaldehyde-modified and 4-hydroxynonenal-modified proteins in the livers of rats with alcoholic liver disease. ( Li, CJ; Lin, RC; Nanji, AA; Siakotos, AN, 1997) |
| "Malondialdehyde (MDA) is an oxidative product of unsaturated fatty acids and is also present in atherosclerotic lesions." | 1.30 | Association of malondialdehyde-acetaldehyde (MAA) adducted proteins with atherosclerotic-induced vascular inflammatory injury. ( Baxter, BT; Duryee, MJ; Hill, GE; Klassen, LW; Miller, JA; Thiele, GM; Tuma, DJ, 1998) |
| "Cortisone treatment of patients suffering from systemic lupus erythematosus and rheumatoid arthritis impressively normalized elevated XOD concentrations in rheumatic sera to those of healthy controls." | 1.29 | Elevated levels of xanthine oxidase in serum of patients with inflammatory and autoimmune rheumatic diseases. ( Miesel, R; Zuber, M, 1993) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (3.13) | 18.7374 |
| 1990's | 6 (18.75) | 18.2507 |
| 2000's | 6 (18.75) | 29.6817 |
| 2010's | 12 (37.50) | 24.3611 |
| 2020's | 7 (21.88) | 2.80 |
| Authors | Studies |
|---|---|
| Duryee, MJ | 4 |
| Clemens, DL | 1 |
| Opperman, PJ | 1 |
| Thiele, GM | 4 |
| Duryee, LM | 1 |
| Garvin, RP | 1 |
| Anderson, DR | 2 |
| Lee, HW | 1 |
| Gu, MJ | 1 |
| Yoo, G | 1 |
| Choi, IW | 1 |
| Lee, SH | 1 |
| Kim, Y | 1 |
| Ha, SK | 1 |
| Niemelä, O | 1 |
| Halkola, AS | 1 |
| Bloigu, A | 1 |
| Bloigu, R | 1 |
| Nivukoski, U | 1 |
| Pohjasniemi, H | 1 |
| Kultti, J | 1 |
| Molina, JC | 1 |
| Guerrero-Morán, JD | 1 |
| González-Espinosa, C | 1 |
| van den Beukel, MD | 1 |
| van Wesemael, TJ | 1 |
| Hoogslag, ATW | 1 |
| Borggreven, NV | 1 |
| Huizinga, TW | 1 |
| van der Helm-van Mil, AH | 1 |
| Toes, RE | 1 |
| van der Woude, D | 1 |
| Trouw, LA | 1 |
| Hunter, CD | 1 |
| England, BR | 1 |
| Fletcher, BS | 1 |
| Daubach, EC | 1 |
| Pospisil, TP | 1 |
| Klassen, LW | 2 |
| Mikuls, TR | 2 |
| Gomes Kjerulf, D | 1 |
| Wang, S | 1 |
| Omer, M | 1 |
| Pathak, A | 1 |
| Subramanian, S | 1 |
| Han, CY | 1 |
| Tang, C | 1 |
| den Hartigh, LJ | 1 |
| Shao, B | 1 |
| Chait, A | 1 |
| Choi, RY | 1 |
| Woo, MJ | 1 |
| Ham, JR | 1 |
| Lee, MK | 1 |
| Tawakol, A | 1 |
| Jaffer, F | 1 |
| Comer, DM | 1 |
| Elborn, JS | 1 |
| Ennis, M | 1 |
| Zambelli, VO | 1 |
| Gross, ER | 1 |
| Chen, CH | 1 |
| Gutierrez, VP | 1 |
| Cury, Y | 1 |
| Mochly-Rosen, D | 1 |
| Antoniak, DT | 1 |
| Feng, CY | 1 |
| Wong, S | 1 |
| Dong, Q | 1 |
| Bruce, J | 1 |
| Mehta, R | 1 |
| Bruce, WR | 1 |
| O'Brien, PJ | 1 |
| Min, JA | 1 |
| Lee, K | 1 |
| Ki, DJ | 1 |
| Buetler, TM | 1 |
| Latado, H | 1 |
| Leclerc, E | 1 |
| Weigle, B | 1 |
| Baumeyer, A | 1 |
| Heizmann, CW | 1 |
| Scholz, G | 1 |
| Maxwell, CR | 1 |
| Spangenberg, RJ | 1 |
| Hoek, JB | 1 |
| Silberstein, SD | 1 |
| Oshinsky, ML | 1 |
| Patsenker, E | 1 |
| Stoll, M | 1 |
| Millonig, G | 1 |
| Agaimy, A | 1 |
| Wissniowski, T | 1 |
| Schneider, V | 1 |
| Mueller, S | 1 |
| Brenneisen, R | 1 |
| Seitz, HK | 1 |
| Ocker, M | 1 |
| Stickel, F | 1 |
| Wyatt, TA | 1 |
| Kharbanda, KK | 1 |
| McCaskill, ML | 1 |
| Tuma, DJ | 2 |
| Yanov, D | 1 |
| DeVasure, J | 1 |
| Sisson, JH | 1 |
| Kawano, T | 1 |
| Matsuse, H | 2 |
| Fukahori, S | 1 |
| Tsuchida, T | 1 |
| Nishino, T | 1 |
| Fukushima, C | 2 |
| Kohno, S | 2 |
| Haas, SL | 1 |
| Ye, W | 1 |
| Löhr, JM | 1 |
| Olszowski, S | 1 |
| Olszowska, E | 1 |
| Kusior, D | 1 |
| Szneler, E | 1 |
| Anderson, MM | 2 |
| Heinecke, JW | 3 |
| Shimoda, T | 1 |
| Sadahiro, A | 1 |
| Singh, AK | 1 |
| Jiang, Y | 1 |
| Benlhabib, E | 1 |
| Gupta, S | 1 |
| Moennikes, O | 1 |
| Vanscheeuwijck, PM | 1 |
| Friedrichs, B | 1 |
| Anskeit, E | 1 |
| Patskan, GJ | 1 |
| Kaelin, RM | 1 |
| Semerjian, A | 1 |
| Center, DM | 1 |
| Bernardo, J | 1 |
| Svegliati-Baroni, G | 1 |
| Baraona, E | 1 |
| Rosman, AS | 1 |
| Lieber, CS | 1 |
| Miesel, R | 1 |
| Zuber, M | 1 |
| Hazen, SL | 2 |
| Hsu, FF | 2 |
| Gaut, JP | 1 |
| Crowley, JR | 1 |
| d'Avignon, A | 1 |
| Li, CJ | 1 |
| Nanji, AA | 1 |
| Siakotos, AN | 1 |
| Lin, RC | 1 |
| Hill, GE | 1 |
| Miller, JA | 1 |
| Baxter, BT | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Oxidative Stress and Surgical Recovery[NCT04732000] | Phase 2 | 21 participants (Actual) | Interventional | 2021-07-01 | Active, not recruiting | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
3 reviews available for acetaldehyde and Inflammation
| Article | Year |
|---|---|
Aldehyde-modified proteins as mediators of early inflammation in atherosclerotic disease.
Topics: Acetaldehyde; Atherosclerosis; Humans; Inflammation; Malondialdehyde | 2015 |
The application of minerals in managing alcohol hangover: a preliminary review.
Topics: Acetaldehyde; Alcohol Drinking; Alcoholic Intoxication; Cytokines; Ethanol; Humans; Inflammation; Mi | 2010 |
Alcohol consumption and digestive tract cancer.
Topics: Acetaldehyde; Alcohol Drinking; Alcoholism; DNA; Ethanol; Gastrointestinal Neoplasms; Gastrointestin | 2012 |
29 other studies available for acetaldehyde and Inflammation
| Article | Year |
|---|---|
Malondialdehyde-Acetaldehyde Modified (MAA) Proteins Differentially Effect the Inflammatory Response in Macrophage, Endothelial Cells and Animal Models of Cardiovascular Disease.
Topics: Acetaldehyde; Animals; Atherosclerosis; Cardiovascular Diseases; Cell Adhesion Molecules; Cells, Cul | 2021 |
Glycolaldehyde induces synergistic effects on vascular inflammation in TNF-α-stimulated vascular smooth muscle cells.
Topics: Acetaldehyde; Animals; Cell Adhesion Molecules; Inflammation; Intercellular Adhesion Molecule-1; Mic | 2022 |
Blood Cell Responses Following Heavy Alcohol Consumption Coincide with Changes in Acute Phase Reactants of Inflammation, Indices of Hemolysis and Immune Responses to Ethanol Metabolites.
Topics: Acetaldehyde; Acute-Phase Proteins; Adult; Alcohol Drinking; Alcoholism; Bilirubin; Biomarkers; Bloo | 2022 |
Alcohol: Immunomodulatory Effects and Cancer.
Topics: Acetaldehyde; Alcohol Drinking; Breast Neoplasms; Ethanol; Female; Humans; Inflammation | 2023 |
Antibodies against advanced glycation end-products and malondialdehyde-acetaldehyde adducts identify a new specific subgroup of hitherto patients with seronegative arthritis with a distinct clinical phenotype and an HLA class II association.
Topics: Acetaldehyde; Arthritis, Rheumatoid; Autoantibodies; Humans; Inflammation; Maillard Reaction; Malond | 2023 |
Immunogenic and inflammatory responses to citrullinated proteins are enhanced following modification with malondialdehyde-acetaldehyde adducts.
Topics: Acetaldehyde; Adjuvants, Immunologic; Animals; Anti-Citrullinated Protein Antibodies; Arthritis, Rhe | 2020 |
Glycation of HDL blunts its anti-inflammatory and cholesterol efflux capacities in vitro, but has no effect in poorly controlled type 1 diabetes subjects.
Topics: Acetaldehyde; Apolipoprotein A-I; Apolipoprotein A-II; Cholesterol; Diabetes Mellitus, Type 1; Gluco | 2020 |
Anti-steatotic and anti-inflammatory effects of Hovenia dulcis Thunb. extracts in chronic alcohol-fed rats.
Topics: Acetaldehyde; Animals; Anti-Inflammatory Agents; Cytochrome P-450 CYP2E1; Down-Regulation; Ethanol; | 2017 |
Imaging the Intersection of Oxidative Stress, Lipids, and Inflammation: Progress Toward Personalized Care of Atherosclerosis.
Topics: Acetaldehyde; Atherosclerosis; Epitopes; Humans; Inflammation; Lipids; Magnetic Resonance Imaging; M | 2018 |
Inflammatory and cytotoxic effects of acrolein, nicotine, acetylaldehyde and cigarette smoke extract on human nasal epithelial cells.
Topics: Acetaldehyde; Acrolein; Cell Death; Cells, Cultured; Epithelial Cells; Humans; Inflammation; Nasal M | 2014 |
Aldehyde dehydrogenase-2 regulates nociception in rodent models of acute inflammatory pain.
Topics: Acetaldehyde; Acute Pain; Aldehyde Dehydrogenase; Aldehyde Dehydrogenase, Mitochondrial; Animals; Be | 2014 |
Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins.
Topics: Acetaldehyde; Animals; Antioxidants; Dose-Response Relationship, Drug; Fructose; Glyoxal; Hepatocyte | 2009 |
Glycolaldehyde-modified β-lactoglobulin AGEs are unable to stimulate inflammatory signaling pathways in RAGE-expressing human cell lines.
Topics: Acetaldehyde; Cell Line; Cytokines; Detergents; Endotoxins; Glycation End Products, Advanced; Humans | 2011 |
Acetate causes alcohol hangover headache in rats.
Topics: Acetaldehyde; Acetates; Alcohol Drinking; Alcoholic Intoxication; Analgesia; Animals; Chronic Diseas | 2010 |
Cannabinoid receptor type I modulates alcohol-induced liver fibrosis.
Topics: Acetaldehyde; Animals; Apoptosis; Cannabinoids; Cell Hypoxia; Cell Proliferation; Collagen; Female; | 2011 |
Malondialdehyde-acetaldehyde-adducted protein inhalation causes lung injury.
Topics: Acetaldehyde; Animals; Chemokines; Ethanol; Female; Humans; Inflammation; Inhalation Exposure; Inter | 2012 |
Acetaldehyde at a low concentration synergistically exacerbates allergic airway inflammation as an endocrine-disrupting chemical and as a volatile organic compound.
Topics: Acetaldehyde; Administration, Intranasal; Animals; Asthma; Bronchoconstriction; Disease Models, Anim | 2012 |
Sulphoacetaldehyde as a product of taurine chloramine peroxidation at site of inflammation.
Topics: Acetaldehyde; Chromatography, High Pressure Liquid; Horseradish Peroxidase; Hydrogen Peroxide; Infla | 2002 |
Production of N(epsilon)-(carboxymethyl)lysine is impaired in mice deficient in NADPH oxidase: a role for phagocyte-derived oxidants in the formation of advanced glycation end products during inflammation.
Topics: Acetaldehyde; Animals; Glycation End Products, Advanced; Hydrogen Peroxide; Inflammation; Lysine; Me | 2003 |
Effects of acetaldehyde on human airway constriction and inflammation.
Topics: Acetaldehyde; Administration, Oral; Aldehyde Dehydrogenase; Aldehyde Dehydrogenase, Mitochondrial; A | 2007 |
Herbal mixtures consisting of puerarin and either polyenylphosphatidylcholine or curcumin provide comprehensive protection against alcohol-related disorders in P rats receiving free choice water and 15% ethanol in pure water.
Topics: Acetaldehyde; Alcohol-Related Disorders; Alcoholism; Animals; Apoptosis; Curcumin; Electroencephalog | 2007 |
Reduced toxicological activity of cigarette smoke by the addition of ammonia magnesium phosphate to the paper of an electrically heated cigarette: subchronic inhalation toxicology.
Topics: Acetaldehyde; Acrolein; Animals; Bronchoalveolar Lavage Fluid; Carbon Monoxide; Carboxyhemoglobin; F | 2008 |
Influence of ethanol on human T-lymphocyte migration.
Topics: Acetaldehyde; Cell Movement; Chemokines, C; Colchicine; Cyclic GMP; Depression, Chemical; Ethanol; H | 1984 |
Collagen-acetaldehyde adducts in alcoholic and nonalcoholic liver diseases.
Topics: Acetaldehyde; Adult; Aged; Analysis of Variance; Animals; Aspartate Aminotransferases; Collagen; gam | 1994 |
Elevated levels of xanthine oxidase in serum of patients with inflammatory and autoimmune rheumatic diseases.
Topics: Acetaldehyde; Acquired Immunodeficiency Syndrome; Autoimmune Diseases; Biomarkers; Cohort Studies; C | 1993 |
Human neutrophils employ the myeloperoxidase-hydrogen peroxide-chloride system to convert hydroxy-amino acids into glycolaldehyde, 2-hydroxypropanal, and acrolein. A mechanism for the generation of highly reactive alpha-hydroxy and alpha,beta-unsaturated
Topics: Acetaldehyde; Acrolein; Aldehydes; Amino Acids; Catalase; Chlorides; Chromatography, High Pressure L | 1997 |
p-Hydroxyphenylacetaldehyde, the major product of L-tyrosine oxidation by the myeloperoxidase-H2O2-chloride system of phagocytes, covalently modifies epsilon-amino groups of protein lysine residues.
Topics: Acetaldehyde; Amino Acids; Animals; Cattle; Chlorides; Chromatography, High Pressure Liquid; Humans; | 1997 |
Acetaldehyde-modified and 4-hydroxynonenal-modified proteins in the livers of rats with alcoholic liver disease.
Topics: Acetaldehyde; Aldehydes; Animals; Antibodies; Corn Oil; Dietary Fats; Fatty Liver; Fish Oils; Humans | 1997 |
Association of malondialdehyde-acetaldehyde (MAA) adducted proteins with atherosclerotic-induced vascular inflammatory injury.
Topics: Acetaldehyde; Animals; Aorta; Arteriosclerosis; Cell Adhesion Molecules; Cell Death; Cells, Cultured | 1998 |