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acebutolol and Multiple Sclerosis

acebutolol has been researched along with Multiple Sclerosis in 1 studies

Acebutolol: A cardioselective beta-1 adrenergic antagonist with little effect on the bronchial receptors. The drug has stabilizing and quinidine-like effects on cardiac rhythm, as well as weak inherent sympathomimetic action.
acebutolol : An ether that is the 2-acetyl-4-(butanoylamino)phenyl ether of the primary hydroxy group of 3-(propan-2-ylamino)propane-1,2-diol.

Multiple Sclerosis: An autoimmune disorder mainly affecting young adults and characterized by destruction of myelin in the central nervous system. Pathologic findings include multiple sharply demarcated areas of demyelination throughout the white matter of the central nervous system. Clinical manifestations include visual loss, extra-ocular movement disorders, paresthesias, loss of sensation, weakness, dysarthria, spasticity, ataxia, and bladder dysfunction. The usual pattern is one of recurrent attacks followed by partial recovery (see MULTIPLE SCLEROSIS, RELAPSING-REMITTING), but acute fulminating and chronic progressive forms (see MULTIPLE SCLEROSIS, CHRONIC PROGRESSIVE) also occur. (Adams et al., Principles of Neurology, 6th ed, p903)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cagliani, R1
Guerini, FR1
Fumagalli, M1
Riva, S1
Agliardi, C1
Galimberti, D1
Pozzoli, U1
Goris, A1
Dubois, B1
Fenoglio, C1
Forni, D1
Sanna, S1
Zara, I1
Pitzalis, M1
Zoledziewska, M1
Cucca, F1
Marini, F1
Comi, GP1
Scarpini, E1
Bresolin, N1
Clerici, M1
Sironi, M1

Other Studies

1 other study available for acebutolol and Multiple Sclerosis

ArticleYear
A trans-specific polymorphism in ZC3HAV1 is maintained by long-standing balancing selection and may confer susceptibility to multiple sclerosis.
    Molecular biology and evolution, 2012, Volume: 29, Issue:6

    Topics: Acebutolol; Animals; Case-Control Studies; Gene-Environment Interaction; Genetic Association Studies

2012