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abt 702 and Hypertrophy

abt 702 has been researched along with Hypertrophy in 1 studies

Hypertrophy: General increase in bulk of a part or organ due to CELL ENLARGEMENT and accumulation of FLUIDS AND SECRETIONS, not due to tumor formation, nor to an increase in the number of cells (HYPERPLASIA).

Research Excerpts

ExcerptRelevanceReference
"There is evidence that extracellular adenosine can attenuate cardiac hypertrophy, but the mechanism by which this occurs is not clear."7.77Adenosine kinase regulation of cardiomyocyte hypertrophy. ( Bache, RJ; Chen, Y; Fassett, JT; Hu, X; Lu, Z; Xu, X; Zhang, P, 2011)
"There is evidence that extracellular adenosine can attenuate cardiac hypertrophy, but the mechanism by which this occurs is not clear."3.77Adenosine kinase regulation of cardiomyocyte hypertrophy. ( Bache, RJ; Chen, Y; Fassett, JT; Hu, X; Lu, Z; Xu, X; Zhang, P, 2011)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fassett, JT1
Hu, X1
Xu, X1
Lu, Z1
Zhang, P1
Chen, Y1
Bache, RJ1

Other Studies

1 other study available for abt 702 and Hypertrophy

ArticleYear
Adenosine kinase regulation of cardiomyocyte hypertrophy.
    American journal of physiology. Heart and circulatory physiology, 2011, Volume: 300, Issue:5

    Topics: Adenosine; Adenosine Kinase; AMP-Activated Protein Kinase Kinases; Animals; Cells, Cultured; Enzyme

2011