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8-cyclopentyl-1,3-dimethylxanthine and Status Epilepticus

8-cyclopentyl-1,3-dimethylxanthine has been researched along with Status Epilepticus in 1 studies

8-cyclopentyl-1,3-dimethylxanthine: prolongs epileptic seizures in rats

Status Epilepticus: A prolonged seizure or seizures repeated frequently enough to prevent recovery between episodes occurring over a period of 20-30 minutes. The most common subtype is generalized tonic-clonic status epilepticus, a potentially fatal condition associated with neuronal injury and respiratory and metabolic dysfunction. Nonconvulsive forms include petit mal status and complex partial status, which may manifest as behavioral disturbances. Simple partial status epilepticus consists of persistent motor, sensory, or autonomic seizures that do not impair cognition (see also EPILEPSIA PARTIALIS CONTINUA). Subclinical status epilepticus generally refers to seizures occurring in an unresponsive or comatose individual in the absence of overt signs of seizure activity. (From N Engl J Med 1998 Apr 2;338(14):970-6; Neurologia 1997 Dec;12 Suppl 6:25-30)

Research Excerpts

ExcerptRelevanceReference
" In the present study we compared neuronal death following status epilepticus (SE) induced in the presence of 8-cyclopentyl-1,3-dimethylxanthine (8-CPT), an A1-adenosine receptor antagonist, with that following SE induced by continuous hippocampal stimulation."3.69Neuronal injury following electrically induced status epilepticus with and without adenosine receptor antagonism. ( Dragunow, M; Young, D, 1995)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (100.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Young, D1
Dragunow, M1

Other Studies

1 other study available for 8-cyclopentyl-1,3-dimethylxanthine and Status Epilepticus

ArticleYear
Neuronal injury following electrically induced status epilepticus with and without adenosine receptor antagonism.
    Experimental neurology, 1995, Volume: 133, Issue:2

    Topics: Animals; Calbindin 1; Calbindins; Cell Death; Electric Stimulation; Electrophysiology; Fibroblast Gr

1995