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7-hydroxy-2-n,n-dipropylaminotetralin and Parkinson Disease, Secondary

7-hydroxy-2-n,n-dipropylaminotetralin has been researched along with Parkinson Disease, Secondary in 2 studies

7-hydroxy-2-N,N-dipropylaminotetralin: RN given refers to cpd without isomeric designation

Parkinson Disease, Secondary: Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (50.00)18.2507
2000's1 (50.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Ueda, H1
Sato, K1
Okumura, F1
Misu, Y1
Bordet, R1
Ridray, S1
Schwartz, JC1
Sokoloff, P1

Other Studies

2 other studies available for 7-hydroxy-2-n,n-dipropylaminotetralin and Parkinson Disease, Secondary

ArticleYear
L-DOPA inhibits spontaneous acetylcholine release from the striatum of experimental Parkinson's model rats.
    Brain research, 1995, Nov-06, Volume: 698, Issue:1-2

    Topics: Acetylcholine; Analysis of Variance; Animals; Corpus Striatum; Disease Models, Animal; Dopamine Agon

1995
Involvement of the direct striatonigral pathway in levodopa-induced sensitization in 6-hydroxydopamine-lesioned rats.
    The European journal of neuroscience, 2000, Volume: 12, Issue:6

    Topics: Animals; Antiparkinson Agents; Benzazepines; Binding, Competitive; Corpus Striatum; Denervation; Diz

2000