7-chlorokynurenic acid has been researched along with Branch Vein Occlusion in 1 studies
7-chlorokynurenic acid: selective antagonist at the glycine modulatory site of the N-methyl-D-aspartate receptor complex; structure given in first source
7-chlorokynurenic acid : A quinolinemonocarboxylic acid that is quinaldic acid which is substituted by a hydroxy group at position 4 and by a chlorine at position 7. It is a potent NMDA glutamate receptor antagonist which antagonizes the strychnine-insensitive glycine site of the NMDA receptor. It also prevents neurodegeneration produced by quinolinic acid.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (100.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Mosinger, JL | 1 |
Price, MT | 1 |
Bai, HY | 1 |
Xiao, H | 1 |
Wozniak, DF | 1 |
Olney, JW | 1 |
1 other study available for 7-chlorokynurenic acid and Branch Vein Occlusion
Article | Year |
---|---|
Blockade of both NMDA and non-NMDA receptors is required for optimal protection against ischemic neuronal degeneration in the in vivo adult mammalian retina.
Topics: Animals; Dose-Response Relationship, Drug; Female; Ischemia; Kynurenic Acid; Light; Nerve Degenerati | 1991 |