Compounds > 6-ketoprostaglandin-f1-alpha

6-ketoprostaglandin-f1-alpha and HIV-Infections

6-ketoprostaglandin-f1-alpha has been researched along with HIV-Infections* in 1 studies

Other Studies

1 other study(ies) available for 6-ketoprostaglandin-f1-alpha and HIV-Infections

Article	Year
Association of lipoprotein(a), prostaglandin I(2)--synthesis stimulating plasma factor, biological half-life of prostaglandin I(2)and high-density lipoproteins in HIV-1 infection of different stages. Prostaglandins, leukotrienes, and essential fatty acids, 2000, Volume: 63, Issue:5 Patients with human immunodeficiency virus show increased atheroembolism and premature arterial events (stroke, myocardial infarction), but no increased venous thromboembolism. This paper describes an association of elevated lipoprotein(a), a decreased prostaglandin I(2)(PGI(2)) synthesis stimulating plasma factor, diminished PGI(2)-stability in plasma and decreased high-density lipoprotein-cholesterol and apolipoprotein A. It is unclear to what extent these biochemical findings represent an acute phase reaction only or a disturbance in the prostaglandin system. Definitely, they are resulting in severe hemostatic imbalance decreasing local PGI(2)-availability with a dramatic reduction in the cytoprotective capacity favouring the onset of premature arterial events seen in some of the patients. Topics: 6-Ketoprostaglandin F1 alpha; Adult; Apolipoproteins A; Body Weight; Cells, Cultured; Cholesterol, HDL; Endothelium, Vascular; Epoprostenol; Female; HIV Infections; HIV Seropositivity; HIV-1; Humans; Lipoprotein(a); Male; Middle Aged; Radioimmunoassay	2000

Article

Year

Association of lipoprotein(a), prostaglandin I(2)--synthesis stimulating plasma factor, biological half-life of prostaglandin I(2)and high-density lipoproteins in HIV-1 infection of different stages.

Prostaglandins, leukotrienes, and essential fatty acids, 2000, Volume: 63, Issue:5

Patients with human immunodeficiency virus show increased atheroembolism and premature arterial events (stroke, myocardial infarction), but no increased venous thromboembolism. This paper describes an association of elevated lipoprotein(a), a decreased prostaglandin I(2)(PGI(2)) synthesis stimulating plasma factor, diminished PGI(2)-stability in plasma and decreased high-density lipoprotein-cholesterol and apolipoprotein A. It is unclear to what extent these biochemical findings represent an acute phase reaction only or a disturbance in the prostaglandin system. Definitely, they are resulting in severe hemostatic imbalance decreasing local PGI(2)-availability with a dramatic reduction in the cytoprotective capacity favouring the onset of premature arterial events seen in some of the patients.

Topics: 6-Ketoprostaglandin F1 alpha; Adult; Apolipoproteins A; Body Weight; Cells, Cultured; Cholesterol, HDL; Endothelium, Vascular; Epoprostenol; Female; HIV Infections; HIV Seropositivity; HIV-1; Humans; Lipoprotein(a); Male; Middle Aged; Radioimmunoassay

2000