6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid and Amyotrophic Lateral Sclerosis

6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid has been researched along with Amyotrophic Lateral Sclerosis in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Chen, K; Liu, Z; Martin, LJ	1

Other Studies

1 other study(ies) available for 6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid and Amyotrophic Lateral Sclerosis

Article	Year
Adult motor neuron apoptosis is mediated by nitric oxide and Fas death receptor linked by DNA damage and p53 activation. The Journal of neuroscience : the official journal of the Society for Neuroscience, 2005, Jul-06, Volume: 25, Issue:27 Topics: Amino Acid Substitution; Amyotrophic Lateral Sclerosis; Animals; Anterior Horn Cells; Antioxidants; Apoptosis; Caspase 8; Caspases; Chromans; Disease Models, Animal; DNA Damage; DNA Glycosylases; DNA Repair; Enzyme Induction; Fas Ligand Protein; fas Receptor; Humans; Male; Membrane Glycoproteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; NADPH Dehydrogenase; Necrosis; Nitric Oxide; Nitric Oxide Synthase Type I; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Oxidative Stress; Peroxynitrous Acid; Point Mutation; Rats; Rats, Sprague-Dawley; Recombinant Fusion Proteins; Sciatic Nerve; Superoxide Dismutase; Superoxide Dismutase-1; Superoxides; Tumor Necrosis Factors; Tumor Suppressor Protein p53	2005

Article

Year

Adult motor neuron apoptosis is mediated by nitric oxide and Fas death receptor linked by DNA damage and p53 activation.

The Journal of neuroscience : the official journal of the Society for Neuroscience, 2005, Jul-06, Volume: 25, Issue:27

Topics: Amino Acid Substitution; Amyotrophic Lateral Sclerosis; Animals; Anterior Horn Cells; Antioxidants; Apoptosis; Caspase 8; Caspases; Chromans; Disease Models, Animal; DNA Damage; DNA Glycosylases; DNA Repair; Enzyme Induction; Fas Ligand Protein; fas Receptor; Humans; Male; Membrane Glycoproteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; NADPH Dehydrogenase; Necrosis; Nitric Oxide; Nitric Oxide Synthase Type I; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Oxidative Stress; Peroxynitrous Acid; Point Mutation; Rats; Rats, Sprague-Dawley; Recombinant Fusion Proteins; Sciatic Nerve; Superoxide Dismutase; Superoxide Dismutase-1; Superoxides; Tumor Necrosis Factors; Tumor Suppressor Protein p53

2005