Page last updated: 2024-08-25

5-hydroxymethylcytosine and Thyroid Neoplasms

5-hydroxymethylcytosine has been researched along with Thyroid Neoplasms in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's2 (66.67)2.80

Authors

AuthorsStudies
Kondo, T; Mochizuki, K; Oishi, N; Vuong, HG1
Astvatsaturyan, K; Fan, X; Lai, J; Peralta-Venturina, M; Seok, JY1
Chen, C; He, N; Hou, P; Ji, M; Jin, T; Liu, S; Liu, W; Qiang, W; Shi, B; Yang, Q1

Other Studies

3 other study(ies) available for 5-hydroxymethylcytosine and Thyroid Neoplasms

ArticleYear
Loss of 5-Hydroxymethylcytosine is an Epigenetic Hallmark of Thyroid Carcinomas with TERT Promoter Mutations.
    Endocrine pathology, 2020, Volume: 31, Issue:4

    Topics: 5-Methylcytosine; Epigenesis, Genetic; Gene Expression Regulation, Neoplastic; Humans; Mutation; Promoter Regions, Genetic; Telomerase; Thyroid Cancer, Papillary; Thyroid Carcinoma, Anaplastic; Thyroid Neoplasms

2020
TROP-2, 5hmC, and IDH1 Expression in Anaplastic Thyroid Carcinoma.
    International journal of surgical pathology, 2021, Volume: 29, Issue:4

    Topics: 5-Methylcytosine; Antibodies, Monoclonal, Humanized; Antigens, Neoplasm; Biomarkers, Tumor; Camptothecin; Cell Adhesion Molecules; Feasibility Studies; Humans; Immunoconjugates; Immunohistochemistry; Isocitrate Dehydrogenase; Molecular Targeted Therapy; Mutation; Patient Selection; Predictive Value of Tests; Prognosis; Thyroid Carcinoma, Anaplastic; Thyroid Gland; Thyroid Neoplasms

2021
PRIMA-1 selectively induces global DNA demethylation in p53 mutant-type thyroid cancer cells.
    Journal of biomedical nanotechnology, 2014, Volume: 10, Issue:7

    Topics: 5-Methylcytosine; Aza Compounds; bcl-2-Associated X Protein; Bridged Bicyclo Compounds, Heterocyclic; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Cyclin-Dependent Kinase Inhibitor p21; Cytosine; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA-Binding Proteins; Down-Regulation; Gene Expression Regulation, Neoplastic; Humans; Mixed Function Oxygenases; Models, Biological; Mutation; Nuclear Proteins; Proto-Oncogene Proteins; Thyroid Neoplasms; Tumor Suppressor Protein p53; Up-Regulation

2014