5-hydroxymethylcytosine has been researched along with Genome Instability in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Cao, Y; Chen, L; Cheng, Y; Jia, J; Jiang, Y; Lai, W; Liu, S; Shi, Y; Tao, Y; Xi, S; Xiao, D; Yan, B | 1 |
Carlton, PM; Hatada, I; Horii, T; Kafer, GR; Li, X; Suetake, I; Tajima, S | 1 |
Abbes, S; Bernard, OA; Cabagnols, X; Della Valle, V; Ishchenko, AA; Mahfoudhi, E; Plo, I; Rameau, P; Saparbaev, M; Secardin, L; Talhaoui, I; Vainchenker, W | 1 |
3 other study(ies) available for 5-hydroxymethylcytosine and Genome Instability
Article | Year |
---|---|
Decrease in Lymphoid Specific Helicase and 5-hydroxymethylcytosine Is Associated with Metastasis and Genome Instability.
Topics: 5-Methylcytosine; Animals; Cell Line, Tumor; Cisplatin; Dioxygenases; DNA Helicases; DNA-Binding Proteins; Drug Resistance, Neoplasm; Gene Expression Regulation, Neoplastic; Genomic Instability; Heterochromatin; Humans; Mice, Nude; MicroRNAs; Neoplasm Metastasis; Protein Binding; Proto-Oncogene Proteins | 2017 |
5-Hydroxymethylcytosine Marks Sites of DNA Damage and Promotes Genome Stability.
Topics: 5-Methylcytosine; Animals; Aphidicolin; Cell Line; CRISPR-Cas Systems; Cytosine; Dioxygenases; DNA Damage; DNA Methylation; DNA Repair; DNA Replication; DNA-Binding Proteins; Epigenesis, Genetic; Genome; Genomic Instability; HeLa Cells; Histones; Human Embryonic Stem Cells; Humans; Mice; Mice, Inbred C57BL; Proto-Oncogene Proteins; Rad51 Recombinase; Tumor Suppressor p53-Binding Protein 1 | 2016 |
TET2-mediated 5-hydroxymethylcytosine induces genetic instability and mutagenesis.
Topics: 5-Methylcytosine; Animals; B-Lymphocytes; Base Sequence; Cell Line; Cytosine; Dioxygenases; DNA Repair; DNA-Binding Proteins; Epigenesis, Genetic; Fibroblasts; Genomic Instability; Humans; Hydroxylation; Megakaryocyte Progenitor Cells; Mice; Mutagenesis; Proto-Oncogene Proteins; S Phase; Thymine DNA Glycosylase; Tumor Suppressor Protein p53 | 2016 |