4-methoxyhonokiol has been researched along with Acute Confusional Senile Dementia in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Ban, JO; Choi, IS; Han, SB; Hong, JT; Jung, JK; Kim, KH; Kim, YH; Lee, HJ; Lee, US; Lee, YK; Oh, KW | 1 |
Choi, DY; Choi, IS; Han, SB; Hong, JT; Hwang, DY; Jeon, YH; Jung, JK; Kim, EH; Kim, KH; Kim, YH; Lee, YH; Lee, YJ; Lee, YK; Oh, KW; Yun, YP | 1 |
Choi, DY; Han, SB; Hong, JT; Kang, JK; Kim, KH; Kim, YH; Lee, BJ; Lee, YJ; Lee, YK; Lee, YM; Nam, SY; Oh, KW; Yun, YW | 1 |
3 other study(ies) available for 4-methoxyhonokiol and Acute Confusional Senile Dementia
Article | Year |
---|---|
4-O-methylhonokiol attenuated β-amyloid-induced memory impairment through reduction of oxidative damages via inactivation of p38 MAP kinase.
Topics: Alzheimer Disease; Amyloid beta-Peptides; Analysis of Variance; Animals; Apoptosis; Astrocytes; Biphenyl Compounds; Cell Survival; Cells, Cultured; Disease Models, Animal; Hippocampus; Lignans; Lipid Peroxidation; Magnolia; Male; Memory Disorders; Mice; Mice, Inbred ICR; Neurons; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Peptide Fragments; Protein Carbonylation; Reactive Oxygen Species | 2011 |
4-O-methylhonokiol attenuated memory impairment through modulation of oxidative damage of enzymes involving amyloid-β generation and accumulation in a mouse model of Alzheimer's disease.
Topics: Alzheimer Disease; Amyloid beta-Peptides; Amyloid beta-Protein Precursor; Amyloid Precursor Protein Secretases; Analysis of Variance; Animals; Avoidance Learning; Biphenyl Compounds; Brain; Circular Dichroism; Disease Models, Animal; Exploratory Behavior; Humans; Lignans; Lipid Peroxidation; Maze Learning; Memory Disorders; Mice; Mice, Transgenic; Neprilysin; Presenilin-2; Protein Carbonylation; Reaction Time | 2011 |
4-O-methylhonokiol prevents memory impairment in the Tg2576 transgenic mice model of Alzheimer's disease via regulation of β-secretase activity.
Topics: Alzheimer Disease; Amyloid beta-Peptides; Amyloid beta-Protein Precursor; Amyloid Precursor Protein Secretases; Analysis of Variance; Animals; Anti-Inflammatory Agents; Avoidance Learning; Biphenyl Compounds; Brain; Disease Models, Animal; Exploratory Behavior; Gene Expression Regulation; Glutathione; Humans; Lignans; Lipid Peroxidation; Maze Learning; Memory Disorders; Mice; Mice, Transgenic; Mutation; Oxidative Stress; Peptide Fragments; Protein Carbonylation; Time Factors | 2012 |