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3-o-methylglucose and Developmental Disabilities

3-o-methylglucose has been researched along with Developmental Disabilities in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (50.00)18.2507
2000's2 (50.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Appleton, DB; Bowling, FG; Burke, CJ; Coman, DJ; De Vivo, DC; McGill, JJ; O'Neil, CM; Pelekanos, JT; Sinclair, KG; Wallace, GB; Wang, D1
De Vivo, DC; Fischbarg, J; Jarjour, IT; Klepper, J; O'Driscoll, KR; Vera, JC; Wang, D1
Brockmann, K; De Vivo, DC; Fischbarg, J; Hanefeld, F; Ho, YY; Korenke, CG; Kranz-Eble, P; Kuang, K; Ma, L; Pascual, JM; von Moers, A; Wang, D; Yang, H1
Behmand, RA; De Vivo, DC; Harik, SI; Jacobson, RI; Ronen, GM; Trifiletti, RR1

Other Studies

4 other study(ies) available for 3-o-methylglucose and Developmental Disabilities

ArticleYear
Seizures, ataxia, developmental delay and the general paediatrician: glucose transporter 1 deficiency syndrome.
    Journal of paediatrics and child health, 2006, Volume: 42, Issue:5

    Topics: 3-O-Methylglucose; Anticonvulsants; Ataxia; Brain Diseases, Metabolic, Inborn; Carbohydrate Metabolism, Inborn Errors; Child; Developmental Disabilities; Diet Therapy; Female; Glucose Transporter Type 1; Humans; Infant; Lumbar Vertebrae; Male; Seizures; Spinal Puncture; Syndrome; Treatment Outcome

2006
Defective glucose transport across brain tissue barriers: a newly recognized neurological syndrome.
    Neurochemical research, 1999, Volume: 24, Issue:4

    Topics: 3-O-Methylglucose; Biological Transport; Brain; Child; Dehydroascorbic Acid; Developmental Disabilities; Erythrocytes; Female; Glucose; Glucose Transporter Type 1; Humans; Immunoblotting; Microcephaly; Monosaccharide Transport Proteins; Mutation; Spasms, Infantile; Syndrome

1999
Autosomal dominant glut-1 deficiency syndrome and familial epilepsy.
    Annals of neurology, 2001, Volume: 50, Issue:4

    Topics: 3-O-Methylglucose; Amino Acid Sequence; Animals; Child; Developmental Disabilities; Epilepsy; Erythrocytes; Family Health; Female; Genes, Dominant; Glucose Transporter Type 1; Humans; Infant, Newborn; Male; Middle Aged; Molecular Sequence Data; Monosaccharide Transport Proteins; Mutation, Missense; Oocytes; Pedigree; Xenopus laevis

2001
Defective glucose transport across the blood-brain barrier as a cause of persistent hypoglycorrhachia, seizures, and developmental delay.
    The New England journal of medicine, 1991, Sep-05, Volume: 325, Issue:10

    Topics: 3-O-Methylglucose; Adult; Biological Transport; Blood Glucose; Blood-Brain Barrier; Child, Preschool; Cytochalasin B; Developmental Disabilities; Erythrocyte Membrane; Female; Glucose; Humans; Infant, Newborn; Male; Methylglucosides; Monosaccharide Transport Proteins; Psychomotor Performance; Seizures

1991