3-o-methylglucose has been researched along with Developmental Disabilities in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (50.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Appleton, DB; Bowling, FG; Burke, CJ; Coman, DJ; De Vivo, DC; McGill, JJ; O'Neil, CM; Pelekanos, JT; Sinclair, KG; Wallace, GB; Wang, D | 1 |
De Vivo, DC; Fischbarg, J; Jarjour, IT; Klepper, J; O'Driscoll, KR; Vera, JC; Wang, D | 1 |
Brockmann, K; De Vivo, DC; Fischbarg, J; Hanefeld, F; Ho, YY; Korenke, CG; Kranz-Eble, P; Kuang, K; Ma, L; Pascual, JM; von Moers, A; Wang, D; Yang, H | 1 |
Behmand, RA; De Vivo, DC; Harik, SI; Jacobson, RI; Ronen, GM; Trifiletti, RR | 1 |
4 other study(ies) available for 3-o-methylglucose and Developmental Disabilities
Article | Year |
---|---|
Seizures, ataxia, developmental delay and the general paediatrician: glucose transporter 1 deficiency syndrome.
Topics: 3-O-Methylglucose; Anticonvulsants; Ataxia; Brain Diseases, Metabolic, Inborn; Carbohydrate Metabolism, Inborn Errors; Child; Developmental Disabilities; Diet Therapy; Female; Glucose Transporter Type 1; Humans; Infant; Lumbar Vertebrae; Male; Seizures; Spinal Puncture; Syndrome; Treatment Outcome | 2006 |
Defective glucose transport across brain tissue barriers: a newly recognized neurological syndrome.
Topics: 3-O-Methylglucose; Biological Transport; Brain; Child; Dehydroascorbic Acid; Developmental Disabilities; Erythrocytes; Female; Glucose; Glucose Transporter Type 1; Humans; Immunoblotting; Microcephaly; Monosaccharide Transport Proteins; Mutation; Spasms, Infantile; Syndrome | 1999 |
Autosomal dominant glut-1 deficiency syndrome and familial epilepsy.
Topics: 3-O-Methylglucose; Amino Acid Sequence; Animals; Child; Developmental Disabilities; Epilepsy; Erythrocytes; Family Health; Female; Genes, Dominant; Glucose Transporter Type 1; Humans; Infant, Newborn; Male; Middle Aged; Molecular Sequence Data; Monosaccharide Transport Proteins; Mutation, Missense; Oocytes; Pedigree; Xenopus laevis | 2001 |
Defective glucose transport across the blood-brain barrier as a cause of persistent hypoglycorrhachia, seizures, and developmental delay.
Topics: 3-O-Methylglucose; Adult; Biological Transport; Blood Glucose; Blood-Brain Barrier; Child, Preschool; Cytochalasin B; Developmental Disabilities; Erythrocyte Membrane; Female; Glucose; Humans; Infant, Newborn; Male; Methylglucosides; Monosaccharide Transport Proteins; Psychomotor Performance; Seizures | 1991 |