3-nitropropionic acid has been researched along with Ischemia in 4 studies
3-nitropropionic acid: succinate dehydrogenase inactivator; biosynthesized by FABACEAE plants from ASPARAGINE
3-nitropropanoic acid : A C-nitro compound that is propanoic acid in which one of the methyl hydrogens has been replaced by a nitro group.
Ischemia: A hypoperfusion of the BLOOD through an organ or tissue caused by a PATHOLOGIC CONSTRICTION or obstruction of its BLOOD VESSELS, or an absence of BLOOD CIRCULATION.
Excerpt | Relevance | Reference |
---|---|---|
"Effect of 3-nitropropionic acid (3-NPA) and ischemia (glucose- and O(2)-free solution) on synaptic transmission in hemisected spinal cord from 4 to 8 day old rats was examined in vitro." | 7.74 | 3-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression. ( Deshpande, SB; Gupta, R, 2008) |
"Memantine was also able to block long-term potentiation recorded from spiny neurons following a brief ischemic episode." | 5.34 | Memantine reduces neuronal dysfunctions triggered by in vitro ischemia and 3-nitropropionic acid. ( Belcastro, V; Calabresi, P; Costa, C; Di Filippo, M; Parnetti, L; Picconi, B; Siliquini, S; Tantucci, M; Tozzi, A, 2007) |
"Effect of 3-nitropropionic acid (3-NPA) and ischemia (glucose- and O(2)-free solution) on synaptic transmission in hemisected spinal cord from 4 to 8 day old rats was examined in vitro." | 3.74 | 3-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression. ( Deshpande, SB; Gupta, R, 2008) |
"The effects of hypoxia (O2-free), aglycemia (glucose-free), ischemia (O2- and glucose-free) and chemical anoxia (by 3-nitropropionic acid; 3-NPA) were evaluated on the synaptic transmission in vitro." | 3.72 | Differential depression of spinal synaptic transmission in vitro by different hypoxic insults. ( Das Gupta, S; Deshpande, SB; Jha, A, 2004) |
"Memantine was also able to block long-term potentiation recorded from spiny neurons following a brief ischemic episode." | 1.34 | Memantine reduces neuronal dysfunctions triggered by in vitro ischemia and 3-nitropropionic acid. ( Belcastro, V; Calabresi, P; Costa, C; Di Filippo, M; Parnetti, L; Picconi, B; Siliquini, S; Tantucci, M; Tozzi, A, 2007) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Gupta, R | 1 |
Deshpande, SB | 2 |
Jha, A | 1 |
Das Gupta, S | 1 |
Lastres-Becker, I | 1 |
Cartmell, T | 1 |
Molina-Holgado, F | 1 |
Tozzi, A | 1 |
Costa, C | 1 |
Di Filippo, M | 1 |
Tantucci, M | 1 |
Siliquini, S | 1 |
Belcastro, V | 1 |
Parnetti, L | 1 |
Picconi, B | 1 |
Calabresi, P | 1 |
4 other studies available for 3-nitropropionic acid and Ischemia
Article | Year |
---|---|
3-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression.
Topics: 2-Amino-5-phosphonovalerate; Action Potentials; Analysis of Variance; Animals; Animals, Newborn; Con | 2008 |
Differential depression of spinal synaptic transmission in vitro by different hypoxic insults.
Topics: Animals; Electrodes; Ganglia, Spinal; Glucose; Hypoxia; Ischemia; Nitro Compounds; Oxygen; Propionat | 2004 |
Endotoxin preconditioning protects neurones from in vitro ischemia: role of endogenous IL-1beta and TNF-alpha.
Topics: Animals; Cell Survival; Cerebellum; Convulsants; Culture Media, Conditioned; Endotoxins; Enzyme-Link | 2006 |
Memantine reduces neuronal dysfunctions triggered by in vitro ischemia and 3-nitropropionic acid.
Topics: Animals; Brain; Convulsants; Dose-Response Relationship, Drug; Drug Interactions; Electric Stimulati | 2007 |