Page last updated: 2024-10-21

3-nitropropionic acid and Ischemia

3-nitropropionic acid has been researched along with Ischemia in 4 studies

3-nitropropionic acid: succinate dehydrogenase inactivator; biosynthesized by FABACEAE plants from ASPARAGINE
3-nitropropanoic acid : A C-nitro compound that is propanoic acid in which one of the methyl hydrogens has been replaced by a nitro group.

Ischemia: A hypoperfusion of the BLOOD through an organ or tissue caused by a PATHOLOGIC CONSTRICTION or obstruction of its BLOOD VESSELS, or an absence of BLOOD CIRCULATION.

Research Excerpts

ExcerptRelevanceReference
"Effect of 3-nitropropionic acid (3-NPA) and ischemia (glucose- and O(2)-free solution) on synaptic transmission in hemisected spinal cord from 4 to 8 day old rats was examined in vitro."7.743-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression. ( Deshpande, SB; Gupta, R, 2008)
"Memantine was also able to block long-term potentiation recorded from spiny neurons following a brief ischemic episode."5.34Memantine reduces neuronal dysfunctions triggered by in vitro ischemia and 3-nitropropionic acid. ( Belcastro, V; Calabresi, P; Costa, C; Di Filippo, M; Parnetti, L; Picconi, B; Siliquini, S; Tantucci, M; Tozzi, A, 2007)
"Effect of 3-nitropropionic acid (3-NPA) and ischemia (glucose- and O(2)-free solution) on synaptic transmission in hemisected spinal cord from 4 to 8 day old rats was examined in vitro."3.743-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression. ( Deshpande, SB; Gupta, R, 2008)
"The effects of hypoxia (O2-free), aglycemia (glucose-free), ischemia (O2- and glucose-free) and chemical anoxia (by 3-nitropropionic acid; 3-NPA) were evaluated on the synaptic transmission in vitro."3.72Differential depression of spinal synaptic transmission in vitro by different hypoxic insults. ( Das Gupta, S; Deshpande, SB; Jha, A, 2004)
"Memantine was also able to block long-term potentiation recorded from spiny neurons following a brief ischemic episode."1.34Memantine reduces neuronal dysfunctions triggered by in vitro ischemia and 3-nitropropionic acid. ( Belcastro, V; Calabresi, P; Costa, C; Di Filippo, M; Parnetti, L; Picconi, B; Siliquini, S; Tantucci, M; Tozzi, A, 2007)

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's4 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Gupta, R1
Deshpande, SB2
Jha, A1
Das Gupta, S1
Lastres-Becker, I1
Cartmell, T1
Molina-Holgado, F1
Tozzi, A1
Costa, C1
Di Filippo, M1
Tantucci, M1
Siliquini, S1
Belcastro, V1
Parnetti, L1
Picconi, B1
Calabresi, P1

Other Studies

4 other studies available for 3-nitropropionic acid and Ischemia

ArticleYear
3-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression.
    Brain research bulletin, 2008, Dec-16, Volume: 77, Issue:6

    Topics: 2-Amino-5-phosphonovalerate; Action Potentials; Analysis of Variance; Animals; Animals, Newborn; Con

2008
Differential depression of spinal synaptic transmission in vitro by different hypoxic insults.
    Indian journal of experimental biology, 2004, Volume: 42, Issue:1

    Topics: Animals; Electrodes; Ganglia, Spinal; Glucose; Hypoxia; Ischemia; Nitro Compounds; Oxygen; Propionat

2004
Endotoxin preconditioning protects neurones from in vitro ischemia: role of endogenous IL-1beta and TNF-alpha.
    Journal of neuroimmunology, 2006, Volume: 173, Issue:1-2

    Topics: Animals; Cell Survival; Cerebellum; Convulsants; Culture Media, Conditioned; Endotoxins; Enzyme-Link

2006
Memantine reduces neuronal dysfunctions triggered by in vitro ischemia and 3-nitropropionic acid.
    Experimental neurology, 2007, Volume: 207, Issue:2

    Topics: Animals; Brain; Convulsants; Dose-Response Relationship, Drug; Drug Interactions; Electric Stimulati

2007