3-nitropropionic acid has been researched along with Hearing Loss, Sensorineural in 7 studies
3-nitropropionic acid: succinate dehydrogenase inactivator; biosynthesized by FABACEAE plants from ASPARAGINE
3-nitropropanoic acid : A C-nitro compound that is propanoic acid in which one of the methyl hydrogens has been replaced by a nitro group.
Hearing Loss, Sensorineural: Hearing loss resulting from damage to the COCHLEA and the sensorineural elements which lie internally beyond the oval and round windows. These elements include the AUDITORY NERVE and its connections in the BRAINSTEM.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Dose-response of the cochlear cells to 3-nitropropionic acid was analyzed in vitro." | 1.39 | Mitochondria toxin-induced acute cochlear cell death indicates cellular activity-correlated energy consumption. ( Poe, D; Pyykkö, I; Yang, S; Zhai, S; Zhang, W; Zhang, Y; Zou, J, 2013) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (28.57) | 29.6817 |
| 2010's | 4 (57.14) | 24.3611 |
| 2020's | 1 (14.29) | 2.80 |
| Authors | Studies |
|---|---|
| Kada, S | 1 |
| Hamaguchi, K | 1 |
| Ito, J | 1 |
| Omori, K | 1 |
| Nakagawa, T | 1 |
| Fujioka, M | 1 |
| Okamoto, Y | 2 |
| Shinden, S | 1 |
| Okano, HJ | 1 |
| Okano, H | 1 |
| Ogawa, K | 2 |
| Matsunaga, T | 4 |
| Fujinami, Y | 3 |
| Mutai, H | 1 |
| Kamiya, K | 3 |
| Mizutari, K | 2 |
| Fujii, M | 3 |
| Zou, J | 2 |
| Li, M | 1 |
| Zhang, Y | 2 |
| Zheng, G | 1 |
| Chen, D | 1 |
| Chen, S | 1 |
| Zheng, H | 1 |
| Zhang, W | 1 |
| Poe, D | 1 |
| Zhai, S | 1 |
| Yang, S | 1 |
| Pyykkö, I | 1 |
| Hoya, N | 1 |
| Kouike, H | 1 |
| Komatsuzaki, R | 1 |
| Kusano, R | 1 |
| Nakagawa, S | 1 |
| Satoh, H | 1 |
7 other studies available for 3-nitropropionic acid and Hearing Loss, Sensorineural
| Article | Year |
|---|---|
Bone Marrow Stromal Cells Accelerate Hearing Recovery via Regeneration or Maintenance of Cochlear Fibrocytes in Mouse Spiral Ligaments.
Topics: Animals; Cochlea; Disease Models, Animal; Evoked Potentials, Auditory, Brain Stem; Female; Hearing L | 2020 |
Pharmacological inhibition of cochlear mitochondrial respiratory chain induces secondary inflammation in the lateral wall: a potential therapeutic target for sensorineural hearing loss.
Topics: Animals; Chemokines; Cochlea; Electron Transport; Female; Gene Expression Profiling; Genome; Hearing | 2014 |
Enhanced expression of C/EBP homologous protein (CHOP) precedes degeneration of fibrocytes in the lateral wall after acute cochlear mitochondrial dysfunction induced by 3-nitropropionic acid.
Topics: Activating Transcription Factor 4; Acute Disease; Animals; Apoptosis; Hearing Loss, Sensorineural; I | 2010 |
Transport augmentation through the blood-inner ear barriers of guinea pigs treated with 3-nitropropionic acid and patients with acute hearing loss, visualized with 3.0 T MRI.
Topics: Adult; Animals; Audiometry; Auditory Threshold; Cochlea; Contrast Media; DNA; DNA Fragmentation; Ear | 2011 |
Mitochondria toxin-induced acute cochlear cell death indicates cellular activity-correlated energy consumption.
Topics: Animals; Auditory Threshold; Cell Death; Cochlea; Disease Models, Animal; DNA Fragmentation; Evoked | 2013 |
Mesenchymal stem cell transplantation accelerates hearing recovery through the repair of injured cochlear fibrocytes.
Topics: Animals; Auditory Threshold; Cochlea; Connexins; Disease Models, Animal; Hearing; Hearing Loss, Sens | 2007 |
Caspase inhibitor facilitates recovery of hearing by protecting the cochlear lateral wall from acute cochlear mitochondrial dysfunction.
Topics: Acoustic Stimulation; Amino Acid Chloromethyl Ketones; Animals; Apoptosis; Auditory Threshold; Caspa | 2008 |