3-nitropropionic acid has been researched along with Cochlear Hearing Loss in 7 studies
3-nitropropionic acid: succinate dehydrogenase inactivator; biosynthesized by FABACEAE plants from ASPARAGINE
3-nitropropanoic acid : A C-nitro compound that is propanoic acid in which one of the methyl hydrogens has been replaced by a nitro group.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Dose-response of the cochlear cells to 3-nitropropionic acid was analyzed in vitro." | 1.39 | Mitochondria toxin-induced acute cochlear cell death indicates cellular activity-correlated energy consumption. ( Poe, D; Pyykkö, I; Yang, S; Zhai, S; Zhang, W; Zhang, Y; Zou, J, 2013) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (28.57) | 29.6817 |
| 2010's | 4 (57.14) | 24.3611 |
| 2020's | 1 (14.29) | 2.80 |
| Authors | Studies |
|---|---|
| Kada, S | 1 |
| Hamaguchi, K | 1 |
| Ito, J | 1 |
| Omori, K | 1 |
| Nakagawa, T | 1 |
| Fujioka, M | 1 |
| Okamoto, Y | 2 |
| Shinden, S | 1 |
| Okano, HJ | 1 |
| Okano, H | 1 |
| Ogawa, K | 2 |
| Matsunaga, T | 4 |
| Fujinami, Y | 3 |
| Mutai, H | 1 |
| Kamiya, K | 3 |
| Mizutari, K | 2 |
| Fujii, M | 3 |
| Zou, J | 2 |
| Li, M | 1 |
| Zhang, Y | 2 |
| Zheng, G | 1 |
| Chen, D | 1 |
| Chen, S | 1 |
| Zheng, H | 1 |
| Zhang, W | 1 |
| Poe, D | 1 |
| Zhai, S | 1 |
| Yang, S | 1 |
| Pyykkö, I | 1 |
| Hoya, N | 1 |
| Kouike, H | 1 |
| Komatsuzaki, R | 1 |
| Kusano, R | 1 |
| Nakagawa, S | 1 |
| Satoh, H | 1 |
7 other studies available for 3-nitropropionic acid and Cochlear Hearing Loss
| Article | Year |
|---|---|
Bone Marrow Stromal Cells Accelerate Hearing Recovery via Regeneration or Maintenance of Cochlear Fibrocytes in Mouse Spiral Ligaments.
Topics: Animals; Cochlea; Disease Models, Animal; Evoked Potentials, Auditory, Brain Stem; Female; Hearing L | 2020 |
Pharmacological inhibition of cochlear mitochondrial respiratory chain induces secondary inflammation in the lateral wall: a potential therapeutic target for sensorineural hearing loss.
Topics: Animals; Chemokines; Cochlea; Electron Transport; Female; Gene Expression Profiling; Genome; Hearing | 2014 |
Enhanced expression of C/EBP homologous protein (CHOP) precedes degeneration of fibrocytes in the lateral wall after acute cochlear mitochondrial dysfunction induced by 3-nitropropionic acid.
Topics: Activating Transcription Factor 4; Acute Disease; Animals; Apoptosis; Hearing Loss, Sensorineural; I | 2010 |
Transport augmentation through the blood-inner ear barriers of guinea pigs treated with 3-nitropropionic acid and patients with acute hearing loss, visualized with 3.0 T MRI.
Topics: Adult; Animals; Audiometry; Auditory Threshold; Cochlea; Contrast Media; DNA; DNA Fragmentation; Ear | 2011 |
Mitochondria toxin-induced acute cochlear cell death indicates cellular activity-correlated energy consumption.
Topics: Animals; Auditory Threshold; Cell Death; Cochlea; Disease Models, Animal; DNA Fragmentation; Evoked | 2013 |
Mesenchymal stem cell transplantation accelerates hearing recovery through the repair of injured cochlear fibrocytes.
Topics: Animals; Auditory Threshold; Cochlea; Connexins; Disease Models, Animal; Hearing; Hearing Loss, Sens | 2007 |
Caspase inhibitor facilitates recovery of hearing by protecting the cochlear lateral wall from acute cochlear mitochondrial dysfunction.
Topics: Acoustic Stimulation; Amino Acid Chloromethyl Ketones; Animals; Apoptosis; Auditory Threshold; Caspa | 2008 |