3-hydroxyflavone has been researched along with Ventricular Dysfunction, Left in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 2 (66.67) | 2.80 |
| Authors | Studies |
|---|---|
| Guo, H; Lin, H; Lin, N; Lu, W; Meng, L; Ni, T; Sun, Z; Zhang, C; Zhang, J | 1 |
| Al-Omary, M; Boyle, AJ; Collins, N; Dib, N; Kovacs, R; McLachlan, G; Moir, S; Schultz, C; Selvanayagam, JB; Selvarajah, A; Sugito, S; Zlotnick, D | 1 |
| Connelly, KA; Edgley, AJ; Kelly, DJ; Khong, FL; Krum, H; Qi, W; Woodman, OL; Zhang, Y | 1 |
1 trial(s) available for 3-hydroxyflavone and Ventricular Dysfunction, Left
| Article | Year |
|---|---|
Calcium/Calmodulin-Dependent Protein Kinase II Delta Inhibition and Ventricular Remodeling After Myocardial Infarction: A Randomized Clinical Trial.
Topics: Aged; Aged, 80 and over; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Double-Blind Method; Female; Flavonols; Humans; Male; Middle Aged; ST Elevation Myocardial Infarction; Ventricular Dysfunction, Left; Ventricular Remodeling | 2021 |
2 other study(ies) available for 3-hydroxyflavone and Ventricular Dysfunction, Left
| Article | Year |
|---|---|
Dihydromyricetin alleviates doxorubicin-induced cardiotoxicity by inhibiting NLRP3 inflammasome through activation of SIRT1.
Topics: Animals; Antibiotics, Antineoplastic; Apoptosis; Cardiotoxicity; Cell Line; Cell Survival; Doxorubicin; Flavonols; Heart; Inflammasomes; Male; Myocardium; Myocytes, Cardiac; NLR Family, Pyrin Domain-Containing 3 Protein; Rats; Rats, Sprague-Dawley; Sirtuin 1; Ventricular Dysfunction, Left | 2020 |
3',4'-Dihydroxyflavonol antioxidant attenuates diastolic dysfunction and cardiac remodeling in streptozotocin-induced diabetic m(Ren2)27 rats.
Topics: Animals; Antioxidants; Autoradiography; Cardiac Catheterization; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Echocardiography; Flavonols; Homozygote; In Situ Hybridization; Male; NADPH Oxidases; Oxidative Stress; Rats; Rats, Transgenic; Reactive Oxygen Species; Ventricular Dysfunction, Left | 2011 |