3-hydroxybutyric acid has been researched along with Poisoning in 5 studies
3-Hydroxybutyric Acid: BUTYRIC ACID substituted in the beta or 3 position. It is one of the ketone bodies produced in the liver.
3-hydroxybutyric acid : A straight-chain 3-hydroxy monocarboxylic acid comprising a butyric acid core with a single hydroxy substituent in the 3- position; a ketone body whose levels are raised during ketosis, used as an energy source by the brain during fasting in humans. Also used to synthesise biodegradable plastics.
Poisoning: Used with drugs, chemicals, and industrial materials for human or animal poisoning, acute or chronic, whether the poisoning is accidental, occupational, suicidal, by medication error, or by environmental exposure.
| Excerpt | Relevance | Reference |
|---|---|---|
| "A patient with acute copper sulfate poisoning was found to have maintained a relatively oxidized hepatic mitochondrial redox state in spite of his being in refractory shock." | 7.69 | Redox state in liver mitochondria in acute copper sulfate poisoning. ( Kobayashi, K; Nakatani, T; Spolter, L, 1994) |
| " Five case reports are presented pertaining to diabetic ketoacidosis in an adult who was not known to suffer from diabetes and in presence of multiple psychotropic substances; fatal flecainide intoxication in a poor metabolizer also presenting an impaired renal function; diabetic ketoacidosis showing severe postmortem changes; primary aldosteronism presented with intracranial hemorrhage and hypothermia showing severe postmortem changes." | 3.78 | Usefulness of postmortem biochemistry in forensic pathology: illustrative case reports. ( Augsburger, M; Lesta, Mdel M; Mangin, P; Palmiere, C; Sabatasso, S; Sporkert, F, 2012) |
| "A patient with acute copper sulfate poisoning was found to have maintained a relatively oxidized hepatic mitochondrial redox state in spite of his being in refractory shock." | 3.69 | Redox state in liver mitochondria in acute copper sulfate poisoning. ( Kobayashi, K; Nakatani, T; Spolter, L, 1994) |
| "Poisoning with potassium cyanide is usually fatal because of the inhibition of cytochrome oxidase." | 3.68 | Changes in the parameters of oxygen metabolism in a clinical course recovering from potassium cyanide. ( Kobayashi, K; Kosugi, Y; Mori, A; Nakatani, T; Tajimi, K, 1993) |
| "Thallium (Tl) is a toxic heavy metal and its exposure to the human body causes physiological and biochemical changes due to its interference with potassium-dependent biological reactions." | 1.37 | Study of acute biochemical effects of thallium toxicity in mouse urine by NMR spectroscopy. ( Bhatnagar, D; Chaturvedi, S; Devi, MM; Khan, AR; Khushu, S; Rana, P; Tripathi, RP; Tyagi, R, 2011) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (40.00) | 18.2507 |
| 2000's | 1 (20.00) | 29.6817 |
| 2010's | 2 (40.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Tyagi, R | 1 |
| Rana, P | 1 |
| Khan, AR | 1 |
| Bhatnagar, D | 1 |
| Devi, MM | 1 |
| Chaturvedi, S | 1 |
| Tripathi, RP | 1 |
| Khushu, S | 1 |
| Palmiere, C | 1 |
| Lesta, Mdel M | 1 |
| Sabatasso, S | 1 |
| Mangin, P | 1 |
| Augsburger, M | 1 |
| Sporkert, F | 1 |
| Smith, SW | 1 |
| Zvosec, DL | 1 |
| Apple, FS | 1 |
| Nakatani, T | 2 |
| Spolter, L | 1 |
| Kobayashi, K | 2 |
| Kosugi, Y | 1 |
| Mori, A | 1 |
| Tajimi, K | 1 |
5 other studies available for 3-hydroxybutyric acid and Poisoning
| Article | Year |
|---|---|
Study of acute biochemical effects of thallium toxicity in mouse urine by NMR spectroscopy.
Topics: 3-Hydroxybutyric Acid; Animals; Choline; Creatinine; Heavy Metal Poisoning; Hippurates; Magnetic Res | 2011 |
Usefulness of postmortem biochemistry in forensic pathology: illustrative case reports.
Topics: 2-Propanol; 3-Hydroxybutyric Acid; Adult; Aged; Anti-Arrhythmia Agents; Benzodiazepines; Biomarkers; | 2012 |
Beta-hydroxybutyrate assay does not cross-react with gamma-hydroxybutyrate.
Topics: 3-Hydroxybutyric Acid; Adult; Blood Chemical Analysis; Cross Reactions; Humans; Male; Poisoning; Sod | 2002 |
Redox state in liver mitochondria in acute copper sulfate poisoning.
Topics: 3-Hydroxybutyric Acid; Adenosine Monophosphate; Adenosine Triphosphate; Animals; Copper; Copper Sulf | 1994 |
Changes in the parameters of oxygen metabolism in a clinical course recovering from potassium cyanide.
Topics: 3-Hydroxybutyric Acid; Acetoacetates; Adult; Antidotes; Electron Transport; Humans; Hydroxybutyrates | 1993 |