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3-hydroxybutyric acid and Cushing Syndrome

3-hydroxybutyric acid has been researched along with Cushing Syndrome in 1 studies

3-Hydroxybutyric Acid: BUTYRIC ACID substituted in the beta or 3 position. It is one of the ketone bodies produced in the liver.
3-hydroxybutyric acid : A straight-chain 3-hydroxy monocarboxylic acid comprising a butyric acid core with a single hydroxy substituent in the 3- position; a ketone body whose levels are raised during ketosis, used as an energy source by the brain during fasting in humans. Also used to synthesise biodegradable plastics.

Cushing Syndrome: A condition caused by prolonged exposure to excess levels of cortisol (HYDROCORTISONE) or other GLUCOCORTICOIDS from endogenous or exogenous sources. It is characterized by upper body OBESITY; OSTEOPOROSIS; HYPERTENSION; DIABETES MELLITUS; HIRSUTISM; AMENORRHEA; and excess body fluid. Endogenous Cushing syndrome or spontaneous hypercortisolism is divided into two groups, those due to an excess of ADRENOCORTICOTROPIN and those that are ACTH-independent.

Research Excerpts

ExcerptRelevanceReference
"Although it is well established that hypercortisolism causes insulin resistance, the mechanisms responsible for impaired insulin action in Cushing's syndrome are unclear."1.30Insulin action and hepatic glucose cycling in Cushing's syndrome. ( Atkinson, AB; Bell, PM; Ennis, C; Harper, R; Heaney, AP; Rooney, DP; Sheridan, B, 1997)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (100.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Heaney, AP1
Harper, R1
Ennis, C1
Rooney, DP1
Sheridan, B1
Atkinson, AB1
Bell, PM1

Other Studies

1 other study available for 3-hydroxybutyric acid and Cushing Syndrome

ArticleYear
Insulin action and hepatic glucose cycling in Cushing's syndrome.
    Clinical endocrinology, 1997, Volume: 46, Issue:6

    Topics: 3-Hydroxybutyric Acid; Adult; Aged; Cushing Syndrome; Fatty Acids, Nonesterified; Female; Glucose; G

1997