3-hydroxybenzylhydrazine has been researched along with Ischemic Attack, Transient in 2 studies
3-hydroxybenzylhydrazine: decarboxylase inhibitor; RN given refers to parent cpd; structure
Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (50.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Misu, Y | 2 |
| Furukawa, N | 2 |
| Arai, N | 2 |
| Miyamae, T | 2 |
| Goshima, Y | 2 |
| Ohshima, E | 1 |
| Suzuki, F | 1 |
| Fujita, K | 1 |
2 other studies available for 3-hydroxybenzylhydrazine and Ischemic Attack, Transient
| Article | Year |
|---|---|
[Is endogenously released DOPA itself an upstream factor for increase in glutamate release and delayed neuronal cell death induced by transient ischemia in rats?].
Topics: Animals; Aromatic Amino Acid Decarboxylase Inhibitors; Cell Death; Corpus Striatum; Dihydroxyphenyla | 1998 |
Endogenously released DOPA is a causal factor for glutamate release and resultant delayed neuronal cell death by transient ischemia in rat striata.
Topics: Animals; Cell Death; Corpus Striatum; Dihydroxyphenylalanine; Dopamine; Enzyme Inhibitors; Glutamic | 2001 |