3-hydroxy-3-methylglutaryl-coenzyme a has been researched along with Lung Neoplasms in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (33.33) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Bai, R; Bode, AM; Chang, X; Dong, Z; Li, X; Liu, K; Ma, W; Ryu, J; Song, Y; Wang, K; Wang, Q; Wang, T; Xia, Q; Zhang, T | 1 |
Chen, Y; Li, W; Li, Y; Liu, H; Wang, Z | 1 |
Berg, D; Clamagirand, C; Couderc, B; Favre, G; Pradines, A; Seronie-Vivien, S; Soula, G | 1 |
3 other study(ies) available for 3-hydroxy-3-methylglutaryl-coenzyme a and Lung Neoplasms
Article | Year |
---|---|
Fluvastatin Inhibits HMG-CoA Reductase and Prevents Non-Small Cell Lung Carcinogenesis.
Topics: Acyl Coenzyme A; Adult; Aged; Animals; Apoptosis; Carcinogenesis; Carcinogens; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Female; Fluvastatin; Gene Knockdown Techniques; HEK293 Cells; Humans; Hydroxymethylglutaryl CoA Reductases; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Lung; Lung Neoplasms; Male; Mevalonic Acid; Mice; Middle Aged; Neoplasms, Experimental; Nitrosamines; RNA, Small Interfering; Xenograft Model Antitumor Assays | 2019 |
Simvastatin prevents proliferation and bone metastases of lung adenocarcinoma in vitro and in vivo.
Topics: Acyl Coenzyme A; Adenocarcinoma; Animals; Apoptosis; Biomarkers, Tumor; Blotting, Western; Bone Neoplasms; Cell Proliferation; Female; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; In Vitro Techniques; Lung Neoplasms; Mice; Mice, Inbred BALB C; Mice, Nude; Real-Time Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Simvastatin; Tumor Cells, Cultured | 2013 |
Reversion of transformed phenotype of human adenocarcinoma A549 cells by expression of 3-hydroxy-3-methylglutaryl coenzyme A reductase complementary DNA.
Topics: Acyl Coenzyme A; Adenocarcinoma; Agar; Animals; Cell Division; Cell Line, Transformed; Cloning, Molecular; Cytoskeleton; DNA, Complementary; Humans; Kinetics; Lung Neoplasms; Mice; Mice, Nude; Oxidoreductases; Phenotype; RNA, Messenger; Transformation, Genetic; Tumor Cells, Cultured | 1995 |