Page last updated: 2024-09-02

3-deoxyglucosone and Innate Inflammatory Response

3-deoxyglucosone has been researched along with Innate Inflammatory Response in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's4 (66.67)24.3611
2020's2 (33.33)2.80

Authors

AuthorsStudies
Auditore, A; Becker, AK; Fleming, T; Messlinger, K; Pischetsrieder, M; Reeh, PW; Sauer, SK1
Carrocera-Pumarino, R; Cepas, V; Collino, M; Collotta, D; Hellwig, M; Henle, T; Manig, F; Mayo, JC; Sainz, RM; Sanmartino, V1
Brouwers, O; de Vos-Houben, JM; Hageman, G; Janssen, BJ; Miyata, T; Niessen, PM; Schalkwijk, CG; Stehouwer, CD; van Nieuwenhoven, F1
Rabbani, N; Thornalley, PJ1
Freund, J; Hellwig, A; Hellwig, M; Henle, T; Herder, C; Hoffmann, B; Krämer, U; Luckhaus, C; Peßler, A; Roden, M; Schikowski, T; Schulte, T; Sugiri, D; Teichert, T; Vierkötter, A; Vossoughi, M1
Bergmans, DC; Schalkwijk, CG; van Bussel, BC; van de Poll, MC1

Reviews

2 review(s) available for 3-deoxyglucosone and Innate Inflammatory Response

ArticleYear
Dicarbonyl stress in cell and tissue dysfunction contributing to ageing and disease.
    Biochemical and biophysical research communications, 2015, Mar-06, Volume: 458, Issue:2

    Topics: Aging; Aldehydes; Cardiovascular Diseases; Deoxyglucose; Diabetes Mellitus; Glyoxal; Humans; Inflammation; Kidney Diseases; Models, Biological; Oxidative Stress; Pyruvaldehyde; Stress, Physiological

2015
Increased Dicarbonyl Stress as a Novel Mechanism of Multi-Organ Failure in Critical Illness.
    International journal of molecular sciences, 2017, Feb-07, Volume: 18, Issue:2

    Topics: Biomarkers; Comorbidity; Critical Care; Critical Illness; Deoxyglucose; Glyoxal; Humans; Hypoxia; Inflammation; Multiple Organ Failure; Pyruvaldehyde; Stress, Physiological

2017

Other Studies

4 other study(ies) available for 3-deoxyglucosone and Innate Inflammatory Response

ArticleYear
Reactive dicarbonyl compounds cause Calcitonin Gene-Related Peptide release and synergize with inflammatory conditions in mouse skin and peritoneum.
    The Journal of biological chemistry, 2020, 05-08, Volume: 295, Issue:19

    Topics: Animals; Bradykinin; Calcitonin Gene-Related Peptide; Deoxyglucose; Drug Interactions; Inflammation; Mice; Mice, Inbred C57BL; Nerve Fibers; Peritoneum; Prostaglandins; Pyruvaldehyde; Skin; Temperature

2020
In Vitro Evaluation of the Toxicological Profile and Oxidative Stress of Relevant Diet-Related Advanced Glycation End Products and Related 1,2-Dicarbonyls.
    Oxidative medicine and cellular longevity, 2021, Volume: 2021

    Topics: Apoptosis; Deoxyglucose; Galactose; Glycation End Products, Advanced; Humans; In Vitro Techniques; Inflammation; Keratinocytes; NF-kappa B; NLR Family, Pyrin Domain-Containing 3 Protein; Oxidative Stress; Pyrones; Reactive Oxygen Species

2021
Mild oxidative damage in the diabetic rat heart is attenuated by glyoxalase-1 overexpression.
    International journal of molecular sciences, 2013, Jul-29, Volume: 14, Issue:8

    Topics: Animals; Chromatography, High Pressure Liquid; Deoxyglucose; Diabetes Mellitus, Experimental; Echocardiography; Fibrosis; Inflammation; Lactoylglutathione Lyase; Lysine; Myocardium; Oxidative Stress; Rats; Tandem Mass Spectrometry; Ventricular Remodeling

2013
Association between Advanced Glycation End Products and Impaired Fasting Glucose: Results from the SALIA Study.
    PloS one, 2015, Volume: 10, Issue:5

    Topics: Aged; Biomarkers; Blood Glucose; Chromatography, Liquid; Cross-Sectional Studies; Deoxyglucose; Fasting; Female; Glucose; Glycation End Products, Advanced; Humans; Inflammation; Insulin; Insulin Resistance; Pilot Projects; Prediabetic State; Tandem Mass Spectrometry

2015