3-deazaneplanocin has been researched along with Disease Exacerbation in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Han, L; Jiang, K; Kang, C; Liu, X; Ning, X; Shi, Z; Zhang, A; Zhang, Q | 1 |
| Chu, DCK; Di Paolo, D; Hardy, T; Leslie, J; Luli, S; Mann, DA; Mann, J; Mendivil, EJ; Moran Salvador, E; Oakley, F; Page, A; Perri, P; Ponzoni, M; Sabater, L; Sharkey, V; Singh, US; Tsukamoto, H; Zeybel, M | 1 |
| Chinnaiyan, AM; He, S; Kato, K; Kuick, R; Liu, Y; Mani, RS; Marquez, VE; Mineishi, S; Mochizuki, K; Nieves, E; Varambally, S; Wang, J; Xie, F; Zhang, Y | 1 |
3 other study(ies) available for 3-deazaneplanocin and Disease Exacerbation
| Article | Year |
|---|---|
DNMT1 and EZH2 mediated methylation silences the microRNA-200b/a/429 gene and promotes tumor progression.
Topics: Adenosine; Animals; Antineoplastic Agents; Azacitidine; Cell Line, Tumor; Decitabine; Disease Progression; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; Enhancer of Zeste Homolog 2 Protein; Gene Expression Regulation, Neoplastic; Gene Silencing; Glioblastoma; Histones; Humans; Mice, Inbred BALB C; Mice, Nude; MicroRNAs; Polycomb Repressive Complex 2; Stomach Neoplasms; Tumor Burden; Xenograft Model Antitumor Assays | 2015 |
A Proof-of-Concept for Epigenetic Therapy of Tissue Fibrosis: Inhibition of Liver Fibrosis Progression by 3-Deazaneplanocin A.
Topics: Adenosine; Animals; Biomarkers; Carbon Tetrachloride; Collagen Type I; Disease Models, Animal; Disease Progression; Epigenesis, Genetic; Gene Expression Profiling; Gene Expression Regulation; Hepatic Stellate Cells; Histone Methyltransferases; Histone-Lysine N-Methyltransferase; Histones; Liver Cirrhosis; Male; Mice; Myofibroblasts | 2017 |
Inhibition of histone methylation arrests ongoing graft-versus-host disease in mice by selectively inducing apoptosis of alloreactive effector T cells.
Topics: Adenosine; Animals; Apoptosis; Cells, Cultured; Disease Progression; Enzyme Inhibitors; Graft vs Host Disease; Histone Methyltransferases; Histone-Lysine N-Methyltransferase; Histones; Isoantigens; Lymphocyte Activation; Methylation; Mice; Mice, Inbred BALB C; Mice, Inbred C3H; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Transgenic; Substrate Specificity; T-Lymphocytes; Up-Regulation | 2012 |