3-amino-6-methyl-5-phenyl-2(1h)-pyridinone has been researched along with Heart Failure in 2 studies
*Heart Failure: A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION. [MeSH]
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 2 (100.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Erhardt, PW | 1 |
Botelho, LH; Herzig, JW; Solaro, RJ | 1 |
1 review(s) available for 3-amino-6-methyl-5-phenyl-2(1h)-pyridinone and Heart Failure
Article | Year |
---|---|
In search of the digitalis replacement.
Topics: 3',5'-Cyclic-AMP Phosphodiesterases; Cardiotonic Agents; Cyclic AMP; Digitalis Glycosides; Heart Failure; Humans; Myocardial Contraction; Receptors, Adrenergic, beta; Structure-Activity Relationship | 1987 |
1 other study(ies) available for 3-amino-6-methyl-5-phenyl-2(1h)-pyridinone and Heart Failure
Article | Year |
---|---|
Myofibrillar Ca++ activation and heart failure--Ca++ sensitization by the cardiotonic agent APP 201-533.
Topics: Animals; Calcium; Cardiotonic Agents; Dogs; Heart Failure; In Vitro Techniques; Liver; Myocardial Contraction; Myofibrils; Protein Kinases; Pyridones; Rats | 1989 |