3,4-methylenedioxyamphetamine has been researched along with Nerve Degeneration in 5 studies
3,4-Methylenedioxyamphetamine: An amphetamine derivative that inhibits uptake of catecholamine neurotransmitters. It is a hallucinogen. It is less toxic than its methylated derivative but in sufficient doses may still destroy serotonergic neurons and has been used for that purpose experimentally.
Nerve Degeneration: Loss of functional activity and trophic degeneration of nerve axons and their terminal arborizations following the destruction of their cells of origin or interruption of their continuity with these cells. The pathology is characteristic of neurodegenerative diseases. Often the process of nerve degeneration is studied in research on neuroanatomical localization and correlation of the neurophysiology of neural pathways.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 4 (80.00) | 18.7374 |
| 1990's | 1 (20.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Schmidt, CJ | 1 |
| Black, CK | 1 |
| Taylor, VL | 1 |
| Battaglia, G | 2 |
| Yeh, SY | 1 |
| De Souza, EB | 2 |
| O'Hearn, E | 1 |
| Kuhar, MJ | 1 |
| Molliver, ME | 1 |
| Gibb, JW | 1 |
| Johnson, M | 1 |
| Hanson, GR | 1 |
| Scallet, AC | 1 |
| Lipe, GW | 1 |
| Ali, SF | 1 |
| Holson, RR | 1 |
| Frith, CH | 1 |
| Slikker, W | 1 |
5 other studies available for 3,4-methylenedioxyamphetamine and Nerve Degeneration
| Article | Year |
|---|---|
L-DOPA potentiation of the serotonergic deficits due to a single administration of 3,4-methylenedioxymethamphetamine, p-chloroamphetamine or methamphetamine to rats.
Topics: 3,4-Methylenedioxyamphetamine; Animals; Brain Chemistry; Cerebral Cortex; Corpus Striatum; Dopamine; | 1991 |
MDMA-induced neurotoxicity: parameters of degeneration and recovery of brain serotonin neurons.
Topics: 3,4-Methylenedioxyamphetamine; Amphetamines; Animals; Biological Transport, Active; Brain; Dose-Resp | 1988 |
Methylenedioxyamphetamine (MDA) and methylenedioxymethamphetamine (MDMA) cause selective ablation of serotonergic axon terminals in forebrain: immunocytochemical evidence for neurotoxicity.
Topics: 3,4-Methylenedioxyamphetamine; Amphetamines; Animals; Axons; Catecholamines; Cell Survival; Dienceph | 1988 |
MK-801 attenuates the methamphetamine induced decreased in tryptophan hydroxylase activity.
Topics: 3,4-Methylenedioxyamphetamine; Animals; Anticonvulsants; Brain; Corpus Striatum; Dibenzocycloheptene | 1989 |
Neuropathological evaluation by combined immunohistochemistry and degeneration-specific methods: application to methylenedioxymethamphetamine.
Topics: 3,4-Methylenedioxyamphetamine; Amphetamines; Animals; Axons; Caudate Nucleus; Immunohistochemistry; | 1988 |