22-hydroxycholesterol has been researched along with Innate Inflammatory Response in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Kanno, M; Kawamoto, M; Ninomiya, Y; Yasuda, T; Yuge, O | 1 |
Burroughs, TJ; Cook, JA; Hake, PW; O'Connor, M; Piraino, G; Teti, D; Zingarelli, B | 1 |
2 other study(ies) available for 22-hydroxycholesterol and Innate Inflammatory Response
Article | Year |
---|---|
Suppression of inducible nitric oxide synthase and cyclooxygenase-2 gene expression by 22(R)-hydroxycholesterol requires de novo protein synthesis in activated macrophages.
Topics: Animals; CCAAT-Enhancer-Binding Proteins; Cell Line; Cells, Cultured; Cyclooxygenase 2; Cyclooxygenase Inhibitors; DNA; DNA-Binding Proteins; Gene Expression Regulation, Enzymologic; Glucocorticoids; Hydroxycholesterols; Inflammation; Ligands; Lipopolysaccharides; Liver X Receptors; Macrophage Activation; Macrophages; Mice; Models, Statistical; NF-kappa B; Nitric Oxide; Nitric Oxide Synthase Type II; Nitrites; Orphan Nuclear Receptors; Protein Binding; Proteins; Receptors, Cytoplasmic and Nuclear; RNA; RNA, Messenger; Time Factors; Transcription Factor AP-1; Transcription, Genetic | 2005 |
Synergistic effect of peroxisome proliferator activated receptor-gamma and liver X receptor-alpha in the regulation of inflammation in macrophages.
Topics: Animals; Anti-Inflammatory Agents; Cells, Cultured; DNA-Binding Proteins; Drug Synergism; Hydrocarbons, Fluorinated; Hydroxycholesterols; Inflammation; Ligands; Lipopolysaccharides; Liver X Receptors; Macrophages; Mice; NF-kappa B; Nitric Oxide; Nitric Oxide Synthase Type II; Orphan Nuclear Receptors; PPAR gamma; Prostaglandin D2; Receptors, Cytoplasmic and Nuclear; Signal Transduction; Sulfonamides; Tumor Necrosis Factor-alpha | 2006 |