2-tert-butylhydroquinone has been researched along with Parkinson Disease in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Cai, X; Chen, L; Hong, F; Kuang, S; Li, Y; Ma, X; Qi, W; Shi, M; Wang, L; Xu, R; Xue, L; Ye, H; Zhang, R | 1 |
Ando, DM; Daub, A; Finkbeiner, S; Finucane, MM; Hwang, V; Lee, AK; Modan, S; Ravisankar, A; Shaby, BA; Skibinski, G | 1 |
Adachi, T; Hara, H; Kuno, S; Ohta, K; Ohta, M | 1 |
Jakel, RJ; Johnson, DA; Johnson, JA; Kern, JT | 1 |
4 other study(ies) available for 2-tert-butylhydroquinone and Parkinson Disease
Article | Year |
---|---|
Identification and optimization of piperine analogues as neuroprotective agents for the treatment of Parkinson's disease via the activation of Nrf2/keap1 pathway.
Topics: Alkaloids; Animals; Apoptosis; Behavior, Animal; Benzodioxoles; Cell Survival; Disease Models, Animal; Dose-Response Relationship, Drug; Kelch-Like ECH-Associated Protein 1; Male; Mice; Mice, Inbred C57BL; Molecular Docking Simulation; Molecular Structure; Neuroprotective Agents; NF-E2-Related Factor 2; Parkinson Disease; PC12 Cells; Piperidines; Polyunsaturated Alkamides; Rats; Structure-Activity Relationship | 2020 |
Nrf2 mitigates LRRK2- and α-synuclein-induced neurodegeneration by modulating proteostasis.
Topics: alpha-Synuclein; Animals; Cerebral Cortex; Genes, Reporter; HEK293 Cells; Humans; Hydroquinones; Inclusion Bodies; Induced Pluripotent Stem Cells; Leucine-Rich Repeat Serine-Threonine Protein Kinase-2; Nerve Tissue Proteins; Neurons; NF-E2-Related Factor 2; Parkinson Disease; Primary Cell Culture; Protein Aggregation, Pathological; Proteostasis; Rats; Recombinant Fusion Proteins; Single-Cell Analysis; Time Factors | 2017 |
Increase of antioxidative potential by tert-butylhydroquinone protects against cell death associated with 6-hydroxydopamine-induced oxidative stress in neuroblastoma SH-SY5Y cells.
Topics: Antioxidants; Cell Death; Cell Line, Tumor; Dose-Response Relationship, Drug; Glutathione; Humans; Hydroquinones; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; Models, Neurological; NAD(P)H Dehydrogenase (Quinone); Neuroblastoma; Neurons; Neuroprotective Agents; Oxidative Stress; Oxidopamine; Parkinson Disease; Reactive Oxygen Species; RNA, Messenger; Transcriptional Activation | 2003 |
Induction of the protective antioxidant response element pathway by 6-hydroxydopamine in vivo and in vitro.
Topics: Animals; Antioxidants; Apoptosis; Astrocytes; Brain Stem; Corpus Striatum; DNA-Binding Proteins; Female; Hydroquinones; Mice; NF-E2-Related Factor 2; Oxidative Stress; Oxidopamine; Parkinson Disease; Receptors, N-Methyl-D-Aspartate; Response Elements; Trans-Activators | 2005 |