2-hydroxychavicol has been researched along with Granulocytic Leukemia, Chronic in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Banerjee, A; Biswas, N; Mahato, SK; Paul, T; Reddy, SVB | 1 |
Bandyopadhyay, S; Biswas, N; Chatterjee, S; Chaudhuri, J; Chaudhuri, U; Chowdhury, AA; Jaisankar, P; Mahato, SK; Manna, A | 1 |
Bandyopadhyay, S; Biswas, N; Chakraborty, JB; Chaudhuri, J; Chaudhuri, U; Choudhury Mukherjee, I; Chowdhury, AA; Ganguly, D; Jaisankar, P; Joshi, K; Konar, A; Mahato, SK; Mandal, L; Manna, A; Pal, BC; Pal, C; Paul, K; Rakshit, S; Roy, S; Shinde, V; Vinayagam, J | 1 |
3 other study(ies) available for 2-hydroxychavicol and Granulocytic Leukemia, Chronic
Article | Year |
---|---|
Hydroxychavicol sensitizes imatinib-resistant chronic myelogenous leukemia cells to TRAIL-induced apoptosis by ROS-mediated IAP downregulation.
Topics: Adaptor Proteins, Signal Transducing; Antineoplastic Agents; Apoptosis; Cell Proliferation; Down-Regulation; Drug Resistance, Neoplasm; Drug Synergism; Eugenol; Gene Expression Regulation, Neoplastic; Humans; Imatinib Mesylate; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Reactive Oxygen Species; TNF-Related Apoptosis-Inducing Ligand; Tumor Cells, Cultured; X-Linked Inhibitor of Apoptosis Protein | 2019 |
Synergistic apoptosis of CML cells by buthionine sulfoximine and hydroxychavicol correlates with activation of AIF and GSH-ROS-JNK-ERK-iNOS pathway.
Topics: Animals; Apoptosis; Apoptosis Inducing Factor; Blotting, Western; Buthionine Sulfoximine; Cell Line, Tumor; Chlorocebus aethiops; Drug Synergism; Eugenol; Extracellular Signal-Regulated MAP Kinases; Glutathione; Hep G2 Cells; Humans; JNK Mitogen-Activated Protein Kinases; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Mice; Microscopy, Confocal; NIH 3T3 Cells; Nitric Oxide; Nitric Oxide Synthase Type II; Reactive Oxygen Species; Signal Transduction; U937 Cells; Vero Cells | 2013 |
Hydroxychavicol, a Piper betle leaf component, induces apoptosis of CML cells through mitochondrial reactive oxygen species-dependent JNK and endothelial nitric oxide synthase activation and overrides imatinib resistance.
Topics: Animals; Antineoplastic Agents; Apoptosis; Benzamides; Blotting, Western; Drug Resistance, Neoplasm; Eugenol; Flow Cytometry; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Leukocytes, Mononuclear; Male; MAP Kinase Kinase 4; Membrane Potential, Mitochondrial; Mice; Mice, Nude; Mice, SCID; Mitochondria; Nitric Oxide; Nitric Oxide Synthase Type III; Phosphorylation; Piper betle; Piperazines; Plant Extracts; Plant Leaves; Pyrimidines; Reactive Oxygen Species; Tumor Cells, Cultured | 2012 |