2-chloroethyl ethyl sulfide has been researched along with Chronic Lung Injury in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Day, BJ; Gould, NS; White, CW | 1 |
Das, SK; Mukherjee, S; Mukhopadhyay, S; Smith, M; Stone, WL | 1 |
Gow, AJ; Laskin, DL; Laskin, JD; Patel-Vayas, K; Shen, J; Sunil, VR | 2 |
4 other study(ies) available for 2-chloroethyl ethyl sulfide and Chronic Lung Injury
Article | Year |
---|---|
A role for mitochondrial oxidative stress in sulfur mustard analog 2-chloroethyl ethyl sulfide-induced lung cell injury and antioxidant protection.
Topics: Antioxidants; Cell Survival; Epithelial Cells; Humans; Lung Injury; Mitochondria; Mitochondrial Membranes; Mustard Gas; Oxidative Stress; Reactive Oxygen Species | 2009 |
Role of MAPK/AP-1 signaling pathway in the protection of CEES-induced lung injury by antioxidant liposome.
Topics: Acetylcysteine; Activating Transcription Factors; Animals; Antidotes; Antioxidants; Blood Proteins; Cell Proliferation; Cyclin D1; Disease Models, Animal; Erythrocytes; Guinea Pigs; Liposomes; Lung; Lung Injury; Male; Mitogen-Activated Protein Kinases; Mustard Gas; Neutrophil Infiltration; Phosphorylation; Proliferating Cell Nuclear Antigen; Proto-Oncogene Proteins c-fos; Proto-Oncogene Proteins c-jun; Pulmonary Eosinophilia; Time Factors; Tocopherols; Transcription Factor AP-1; Tumor Necrosis Factor-alpha | 2009 |
Role of TNFR1 in lung injury and altered lung function induced by the model sulfur mustard vesicant, 2-chloroethyl ethyl sulfide.
Topics: Animals; Bronchoalveolar Lavage Fluid; Irritants; Lung; Lung Injury; Male; Mice; Mice, Knockout; Mustard Gas; Oxidative Stress; Receptors, Tumor Necrosis Factor, Type I; Tumor Necrosis Factor-alpha | 2011 |
Role of reactive nitrogen species generated via inducible nitric oxide synthase in vesicant-induced lung injury, inflammation and altered lung functioning.
Topics: Acute-Phase Proteins; Animals; Chemical Warfare Agents; Female; Gene Expression Regulation; Inflammation; Lipocalin-2; Lipocalins; Lung; Lung Injury; Mice; Mice, Inbred C57BL; Mice, Knockout; Mustard Gas; Nitric Oxide Synthase Type II; Oncogene Proteins; Oxidative Stress; Reactive Nitrogen Species; Time Factors; Tumor Necrosis Factor-alpha | 2012 |