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2-chloro-n(6)cyclopentyladenosine and Acute Kidney Injury

2-chloro-n(6)cyclopentyladenosine has been researched along with Acute Kidney Injury in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Brown, KM; D'Agati, VD; Greene, RW; Ham, A; Kim, JY; Kim, M; Lee, HT1
Brown, KM; D'Agati, VD; Haase, VH; Kim, JY; Kim, M; Lee, HT; Park, SW1
Emala, CW; Lee, HT; Nasr, SH; Schnermann, J; Xu, H1

Other Studies

3 other study(ies) available for 2-chloro-n(6)cyclopentyladenosine and Acute Kidney Injury

ArticleYear
IL-11 is required for A1 adenosine receptor-mediated protection against ischemic AKI.
    Journal of the American Society of Nephrology : JASN, 2013, Volume: 24, Issue:10

    Topics: Acute Kidney Injury; Adenosine; Adenosine A1 Receptor Agonists; Animals; Cell Line; Humans; Interleukin-11; Male; Mice; Mice, Inbred C57BL; Receptor, Adenosine A1; Reperfusion Injury

2013
Proximal tubule sphingosine kinase-1 has a critical role in A1 adenosine receptor-mediated renal protection from ischemia.
    Kidney international, 2012, Volume: 82, Issue:8

    Topics: Acute Kidney Injury; Adenosine; Adenosine A1 Receptor Agonists; Animals; Kidney; Kidney Tubules, Proximal; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Models, Statistical; Phosphotransferases (Alcohol Group Acceptor); Receptor, Adenosine A1; Receptors, Lysosphingolipid; Reperfusion Injury

2012
A1 adenosine receptor knockout mice exhibit increased renal injury following ischemia and reperfusion.
    American journal of physiology. Renal physiology, 2004, Volume: 286, Issue:2

    Topics: Acute Kidney Injury; Adenosine; Adenosine A1 Receptor Antagonists; Animals; Apoptosis; Biomarkers; Genotype; In Situ Nick-End Labeling; Intercellular Adhesion Molecule-1; Interleukin-1; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Necrosis; Neutrophils; Receptor, Adenosine A1; Reperfusion Injury; RNA, Messenger; Tumor Necrosis Factor-alpha; Xanthines

2004