2-aminofluorene has been researched along with Leukemia in 2 studies
Leukemia: A progressive, malignant disease of the blood-forming organs, characterized by distorted proliferation and development of leukocytes and their precursors in the blood and bone marrow. Leukemias were originally termed acute or chronic based on life expectancy but now are classified according to cellular maturity. Acute leukemias consist of predominately immature cells; chronic leukemias are composed of more mature cells. (From The Merck Manual, 2006)
Excerpt | Relevance | Reference |
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" The purpose of this present study is to evaluate the question of whether luteolin could affect arylamine N-acetyltransferase (NAT) activity and DNA-2-aminofluorene adduct formation in human (HL-60) and mouse (L1210) leukemia cells." | 7.71 | Luteolin-inhibited arylamine N-acetyltransferase activity and DNA-2-aminofluorene adduct in human and mouse leukemia cells. ( Chung, JG; Hung, CF; Li, YC; Lin, JP; Yeh, FT, 2001) |
" In this study, paclitaxel was selected to determine the inhibition of arylamine N-acetyltransferase activity, gene expression (NAT mRNA) and DNA-2-aminofluorene adduct formation in human leukemia HL-60 cell line." | 7.71 | The effect of paclitaxel on gene expression and activity of arylamine N-acetyltransferase and DNA-2-aminofluorene adduct formation in human leukemia HL-60 cells. ( Chou, MC; Chung, JG; Ho, HC; Hsia, TC; Hsiao, YM; Lin, KL; Lu, KH; Yang, CC, 2002) |
" The purpose of this present study is to evaluate the question of whether luteolin could affect arylamine N-acetyltransferase (NAT) activity and DNA-2-aminofluorene adduct formation in human (HL-60) and mouse (L1210) leukemia cells." | 3.71 | Luteolin-inhibited arylamine N-acetyltransferase activity and DNA-2-aminofluorene adduct in human and mouse leukemia cells. ( Chung, JG; Hung, CF; Li, YC; Lin, JP; Yeh, FT, 2001) |
" In this study, paclitaxel was selected to determine the inhibition of arylamine N-acetyltransferase activity, gene expression (NAT mRNA) and DNA-2-aminofluorene adduct formation in human leukemia HL-60 cell line." | 3.71 | The effect of paclitaxel on gene expression and activity of arylamine N-acetyltransferase and DNA-2-aminofluorene adduct formation in human leukemia HL-60 cells. ( Chou, MC; Chung, JG; Ho, HC; Hsia, TC; Hsiao, YM; Lin, KL; Lu, KH; Yang, CC, 2002) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Li, YC | 1 |
Hung, CF | 1 |
Yeh, FT | 1 |
Lin, JP | 1 |
Chung, JG | 2 |
Lu, KH | 1 |
Lin, KL | 1 |
Yang, CC | 1 |
Hsia, TC | 1 |
Hsiao, YM | 1 |
Chou, MC | 1 |
Ho, HC | 1 |
2 other studies available for 2-aminofluorene and Leukemia
Article | Year |
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Luteolin-inhibited arylamine N-acetyltransferase activity and DNA-2-aminofluorene adduct in human and mouse leukemia cells.
Topics: 2-Acetylaminofluorene; Acetylation; Animals; Arylamine N-Acetyltransferase; Cytosol; DNA Adducts; Do | 2001 |
The effect of paclitaxel on gene expression and activity of arylamine N-acetyltransferase and DNA-2-aminofluorene adduct formation in human leukemia HL-60 cells.
Topics: Acetylation; Animals; Antineoplastic Agents, Phytogenic; Arylamine N-Acetyltransferase; Carcinogens; | 2002 |