2-aminoethoxydiphenyl borate has been researched along with Liver Failure, Acute in 2 studies
2-aminoethoxydiphenyl borate: is a novel membrane-penetrable modulator and transient receptor potential channel blocker; structure in first source; do not confuse with 2-APB cpd
2-aminoethoxydiphenylborane : An organoboron compound that is diphenylborane in which the borane hydrogen is replaced by a 2-aminoethoxy group.
Liver Failure, Acute: A form of rapid-onset LIVER FAILURE, also known as fulminant hepatic failure, caused by severe liver injury or massive loss of HEPATOCYTES. It is characterized by sudden development of liver dysfunction and JAUNDICE. Acute liver failure may progress to exhibit cerebral dysfunction even HEPATIC COMA depending on the etiology that includes hepatic ISCHEMIA, drug toxicity, malignant infiltration, and viral hepatitis such as post-transfusion HEPATITIS B and HEPATITIS C.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The mechanism of renal failure during fulminant hepatic failure (FHF) or end-stage of liver disease is not fully understood." | 1.40 | Tumor necrosis factor-alpha-induced reduction of glomerular filtration rate in rats with fulminant hepatic failure. ( Liu, P; Sun, CM; Wang, DL; Wang, HT; Wang, JB; Wang, ZH; Wen, Y; Wu, J; Zhao, YT, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Wang, JB | 1 |
| Wang, DL | 1 |
| Wang, HT | 1 |
| Wang, ZH | 1 |
| Wen, Y | 1 |
| Sun, CM | 1 |
| Zhao, YT | 1 |
| Wu, J | 1 |
| Liu, P | 1 |
| Patel, SJ | 1 |
| Milwid, JM | 1 |
| King, KR | 1 |
| Bohr, S | 1 |
| Iracheta-Vellve, A | 1 |
| Iracheta-Velle, A | 1 |
| Li, M | 1 |
| Vitalo, A | 1 |
| Parekkadan, B | 1 |
| Jindal, R | 1 |
| Yarmush, ML | 1 |
2 other studies available for 2-aminoethoxydiphenyl borate and Liver Failure, Acute
| Article | Year |
|---|---|
Tumor necrosis factor-alpha-induced reduction of glomerular filtration rate in rats with fulminant hepatic failure.
Topics: Animals; Antibodies; Blotting, Western; Boron Compounds; Calcium; Galactosamine; Gene Expression; Gl | 2014 |
Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure.
Topics: Acetaminophen; Animals; Boron Compounds; Chemical and Drug Induced Liver Injury; Connexins; Gap Junc | 2012 |