Page last updated: 2024-10-17

2-aminoadipic acid and Carbon Tetrachloride Poisoning

2-aminoadipic acid has been researched along with Carbon Tetrachloride Poisoning in 1 studies

2-Aminoadipic Acid: A metabolite in the principal biochemical pathway of lysine. It antagonizes neuroexcitatory activity modulated by the glutamate receptor, N-METHYL-D-ASPARTATE; (NMDA).
2-aminoadipic acid : An alpha-amino acid that is adipic acid bearing a single amino substituent at position 2. An intermediate in the formation of lysine.

Carbon Tetrachloride Poisoning: Poisoning that results from ingestion, injection, inhalation, or skin absorption of CARBON TETRACHLORIDE.

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Umeda, H1
Takeuchi, M1
Suyama, K1

Other Studies

1 other study available for 2-aminoadipic acid and Carbon Tetrachloride Poisoning

ArticleYear
Two new elastin cross-links having pyridine skeleton. Implication of ammonia in elastin cross-linking in vivo.
    The Journal of biological chemistry, 2001, Apr-20, Volume: 276, Issue:16

    Topics: 2-Aminoadipic Acid; Aging; Amino Acids; Ammonium Chloride; Animals; Aorta; Carbon Tetrachloride Pois

2001