Page last updated: 2024-10-22

2-amino-4-phosphonobutyric acid and Parkinson Disease, Secondary

2-amino-4-phosphonobutyric acid has been researched along with Parkinson Disease, Secondary in 1 studies

2-amino-4-phosphonobutyric acid: glutamate antagonist in locust muscle; structure; do not confuse with L-AP4, which is the propionic acid version

Parkinson Disease, Secondary: Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cuomo, D1
Martella, G1
Barabino, E1
Platania, P1
Vita, D1
Madeo, G1
Selvam, C1
Goudet, C1
Oueslati, N1
Pin, JP1
Acher, F1
Pisani, A1
Beurrier, C1
Melon, C1
Kerkerian-Le Goff, L1
Gubellini, P1

Other Studies

1 other study available for 2-amino-4-phosphonobutyric acid and Parkinson Disease, Secondary

ArticleYear
Metabotropic glutamate receptor subtype 4 selectively modulates both glutamate and GABA transmission in the striatum: implications for Parkinson's disease treatment.
    Journal of neurochemistry, 2009, Volume: 109, Issue:4

    Topics: Aminobutyrates; Animals; Antiparkinson Agents; Dose-Response Relationship, Drug; Electrophysiology;

2009