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2-amino-4-phosphonobutyric acid and Nerve Degeneration

2-amino-4-phosphonobutyric acid has been researched along with Nerve Degeneration in 3 studies

2-amino-4-phosphonobutyric acid: glutamate antagonist in locust muscle; structure; do not confuse with L-AP4, which is the propionic acid version

Nerve Degeneration: Loss of functional activity and trophic degeneration of nerve axons and their terminal arborizations following the destruction of their cells of origin or interruption of their continuity with these cells. The pathology is characteristic of neurodegenerative diseases. Often the process of nerve degeneration is studied in research on neuroanatomical localization and correlation of the neurophysiology of neural pathways.

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's2 (66.67)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Zhou, F1
Hongmin, B1
Xiang, Z1
Enyu, L1
Copani, A1
Bruno, V2
Battaglia, G2
Leanza, G1
Pellitteri, R1
Russo, A1
Stanzani, S1
Nicoletti, F2
Ksiazek, I1
van der Putten, H1
Catania, MV1
Giuffrida, R1
Lukic, S1
Leonhardt, T1
Inderbitzin, W1
Gasparini, F1
Kuhn, R1
Hampson, DR1
Flor, PJ1

Other Studies

3 other studies available for 2-amino-4-phosphonobutyric acid and Nerve Degeneration

ArticleYear
Changes of mGluR4 and the effects of its specific agonist L-AP4 in a rodent model of diffuse brain injury.
    Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 2003, Volume: 10, Issue:6

    Topics: Aminobutyrates; Animals; Behavior, Animal; Brain; Brain Injuries; Cell Count; Cognition Disorders; D

2003
Activation of metabotropic glutamate receptors protects cultured neurons against apoptosis induced by beta-amyloid peptide.
    Molecular pharmacology, 1995, Volume: 47, Issue:5

    Topics: Aminobutyrates; Amyloid beta-Peptides; Animals; Apoptosis; Benzoates; Calcium Channels; Cells, Cultu

1995
Selective activation of mGlu4 metabotropic glutamate receptors is protective against excitotoxic neuronal death.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2000, Sep-01, Volume: 20, Issue:17

    Topics: Aminobutyrates; Animals; Cells, Cultured; Cerebral Cortex; Excitatory Amino Acid Agonists; Glutamic

2000