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2-amino-1-methyl-6-phenylimidazo(4,5-b)pyridine and Kidney Neoplasms

2-amino-1-methyl-6-phenylimidazo(4,5-b)pyridine has been researched along with Kidney Neoplasms in 2 studies

2-amino-1-methyl-6-phenylimidazo(4,5-b)pyridine: mutagen from fried ground beef; structure given in first source
PhIP : An imidazopyridine that is 1H--imidazo[4,5-b]pyridine which is substituted at positions 1, 2, and 6 by methyl, amino, and phenyl groups, respectively. It is the most abundant of the mutagenic heterocyclic amines found in cooked meat and fish.

Kidney Neoplasms: Tumors or cancers of the KIDNEY.

Research Excerpts

ExcerptRelevanceReference
"These associations may be modified by genetic susceptibility to RCC."1.43Gene-environment interaction of genome-wide association study-identified susceptibility loci and meat-cooking mutagens in the etiology of renal cell carcinoma. ( Daniel, CR; Karam, JA; Matin, SF; Melkonian, SC; Tannir, NM; Wood, CG; Wu, X; Ye, Y, 2016)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Yeh, IJ1
Chen, SC1
Yen, MC1
Wu, YH1
Hung, CH1
Kuo, PL1
Melkonian, SC1
Daniel, CR1
Ye, Y1
Tannir, NM1
Karam, JA1
Matin, SF1
Wood, CG1
Wu, X1

Other Studies

2 other studies available for 2-amino-1-methyl-6-phenylimidazo(4,5-b)pyridine and Kidney Neoplasms

ArticleYear
6-Shogaol Suppresses 2-Amino-1-Methyl-6-Phenylimidazo [4,5-b] Pyridine (PhIP)-Induced Human 786-O Renal Cell Carcinoma Osteoclastogenic Activity and Metastatic Potential.
    Nutrients, 2019, Sep-28, Volume: 11, Issue:10

    Topics: Carcinoma, Renal Cell; Catechols; Cell Line, Tumor; Humans; Imidazoles; Kidney Neoplasms; Neoplasm M

2019
Gene-environment interaction of genome-wide association study-identified susceptibility loci and meat-cooking mutagens in the etiology of renal cell carcinoma.
    Cancer, 2016, Jan-01, Volume: 122, Issue:1

    Topics: Carcinoma, Renal Cell; Case-Control Studies; Disease Susceptibility; Female; Gene-Environment Intera

2016