2,8-dihydroxyadenine has been researched along with Inflammation in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (50.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Boor, P; Buhl, EM; Dehairs, J; Jankowski, J; Klinkhammer, BM; Krueger, K; Lebherz, C; Lehrke, M; Marx, N; Moellmann, J; Noels, H; Swinnen, JV; Wong, DWL | 1 |
| Boivin, G; Chen, J; Davies, PM; Engle, SJ; Sahota, A; Simmonds, HA; Stambrook, PJ; Stockelman, MG; Tischfield, JA; Ying, MY; Yum, MN | 1 |
2 other study(ies) available for 2,8-dihydroxyadenine and Inflammation
| Article | Year |
|---|---|
2,8-Dihydroxyadenine-induced nephropathy causes hexosylceramide accumulation with increased mTOR signaling, reduced levels of protective SirT3 expression and impaired renal mitochondrial function.
Topics: Adenine; Animals; Fibrosis; Inflammation; Kidney; Mice; Mice, Inbred C57BL; Mitochondria; Renal Insufficiency, Chronic; Sirtuin 3; TOR Serine-Threonine Kinases | 2024 |
Adenine phosphoribosyltransferase-deficient mice develop 2,8-dihydroxyadenine nephrolithiasis.
Topics: Adenine; Adenine Phosphoribosyltransferase; Alleles; Animals; Erythrocytes; Fibrosis; Homozygote; Humans; Inflammation; Kidney; Kidney Calculi; Mice; Mice, Knockout; Necrosis; Recombination, Genetic; Restriction Mapping; Stem Cells | 1996 |