2,5-dihydroxybenzoic acid has been researched along with Hyperglycemia in 3 studies
2,5-dihydroxybenzoic acid: RN given refers to parent cpd; a oxidative product of saligenin
2,5-dihydroxybenzoic acid : A dihydroxybenzoic acid having the two hydroxy groups at the 2- and 5-positions.
Hyperglycemia: Abnormally high BLOOD GLUCOSE level.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Acarbose reduces myocardial infarct size by opening mitochondrial KATP channels, which may be related to the prevention of postprandial hyperglycemia and hydroxyl radical production." | 7.75 | Acarbose reduces myocardial infarct size by preventing postprandial hyperglycemia and hydroxyl radical production and opening mitochondrial KATP channels in rabbits. ( Bao, N; Fujiwara, H; Fujiwara, T; Iwasa, M; Kawamura, I; Kobayashi, H; Minatoguchi, S; Nishigaki, K; Sumi, S; Takemura, G; Yamada, Y; Yasuda, S; Zhang, Z, 2009) |
| "Acarbose reduces myocardial infarct size by opening mitochondrial KATP channels, which may be related to the prevention of postprandial hyperglycemia and hydroxyl radical production." | 3.75 | Acarbose reduces myocardial infarct size by preventing postprandial hyperglycemia and hydroxyl radical production and opening mitochondrial KATP channels in rabbits. ( Bao, N; Fujiwara, H; Fujiwara, T; Iwasa, M; Kawamura, I; Kobayashi, H; Minatoguchi, S; Nishigaki, K; Sumi, S; Takemura, G; Yamada, Y; Yasuda, S; Zhang, Z, 2009) |
| "Preexisting hyperglycemia is associated with enhanced reperfusion injury in the postischemic rat brain." | 1.30 | Hydroxyl radical formation in hyperglycemic rats during middle cerebral artery occlusion/reperfusion. ( Huang, NC; Quast, MJ; Wei, J, 1997) |
| "Preischemic hyperglycemia is known to aggravate brain damage resulting from transient ischemia." | 1.30 | Production of hydroxyl free radical by brain tissues in hyperglycemic rats subjected to transient forebrain ischemia. ( Floyd, RA; He, QP; Li, PA; Liu, GJ; Siesjö, BK, 1999) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (66.67) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Minatoguchi, S | 1 |
| Zhang, Z | 1 |
| Bao, N | 1 |
| Kobayashi, H | 1 |
| Yasuda, S | 1 |
| Iwasa, M | 1 |
| Sumi, S | 1 |
| Kawamura, I | 1 |
| Yamada, Y | 1 |
| Nishigaki, K | 1 |
| Takemura, G | 1 |
| Fujiwara, T | 1 |
| Fujiwara, H | 1 |
| Wei, J | 1 |
| Huang, NC | 1 |
| Quast, MJ | 1 |
| Li, PA | 1 |
| Liu, GJ | 1 |
| He, QP | 1 |
| Floyd, RA | 1 |
| Siesjö, BK | 1 |
3 other studies available for 2,5-dihydroxybenzoic acid and Hyperglycemia
| Article | Year |
|---|---|
Acarbose reduces myocardial infarct size by preventing postprandial hyperglycemia and hydroxyl radical production and opening mitochondrial KATP channels in rabbits.
Topics: Acarbose; Animals; Blood Glucose; Catechols; Enzyme Inhibitors; Gentisates; Hydroxybenzoates; Hydrox | 2009 |
Hydroxyl radical formation in hyperglycemic rats during middle cerebral artery occlusion/reperfusion.
Topics: Animals; Arterial Occlusive Diseases; Cerebral Arteries; Cerebral Infarction; Enzyme Inhibitors; Gen | 1997 |
Production of hydroxyl free radical by brain tissues in hyperglycemic rats subjected to transient forebrain ischemia.
Topics: Animals; Brain; Brain Injuries; Brain Ischemia; Gentisates; Hydroxybenzoates; Hydroxyl Radical; Hype | 1999 |