2,5-bis(3,4,5-trimethoxyphenyl)tetrahydrofuran has been researched along with Bright Disease in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 3 (100.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Benigni, A; Bertani, T; Livio, M; Macconi, D; Morigi, M; Orisio, S; Patrono, C; Perico, N; Remuzzi, G; Ubiali, A | 1 |
| Benigni, A; Livio, M; Macconi, D; Morigi, M; Orisio, S; Patrono, C; Remuzzi, G | 1 |
| Bertani, T; Bisogno, G; Livio, M; Macconi, D; Morigi, M; Patrono, C; Remuzzi, G | 1 |
3 other study(ies) available for 2,5-bis(3,4,5-trimethoxyphenyl)tetrahydrofuran and Bright Disease
| Article | Year |
|---|---|
Enhanced glomerular thromboxane A2 mediates some pathophysiologic effect of platelet-activating factor in rabbit nephrotoxic nephritis: evidence from biochemical measurements and inhibitor trials.
Topics: Animals; Arachidonic Acid; Arachidonic Acids; Furans; Glomerulonephritis; Kidney; Kidney Glomerulus; Male; Platelet Activating Factor; Platelet Aggregation; Platelet Membrane Glycoproteins; Prostaglandins; Rabbits; Receptors, Cell Surface; Receptors, G-Protein-Coupled; Thromboxane A2; Thromboxane-A Synthase | 1989 |
A platelet-activating factor receptor antagonist protects against renal function deterioration in nephrotoxic nephritis.
Topics: Animals; Furans; Glomerular Filtration Rate; Glomerulonephritis; Male; Platelet Activating Factor; Platelet Membrane Glycoproteins; Proteinuria; Rabbits; Receptors, Cell Surface; Receptors, G-Protein-Coupled | 1987 |
Platelet activating factor (PAF) as a mediator of injury in nephrotoxic nephritis.
Topics: Animals; Creatinine; Furans; Glomerulonephritis; Kidney Function Tests; Kidney Glomerulus; Male; Platelet Activating Factor; Platelet Membrane Glycoproteins; Proteinuria; Rabbits; Receptors, Cell Surface; Receptors, G-Protein-Coupled | 1987 |