15-ketoprostaglandin-f2alpha has been researched along with Dysmenorrhea* in 1 studies
1 other study(ies) available for 15-ketoprostaglandin-f2alpha and Dysmenorrhea
| Article | Year |
|---|---|
Does aerobic exercise induced-analgesia occur through hormone and inflammatory cytokine-mediated mechanisms in primary dysmenorrhea?
The popular accepted explanation for the pathogenesis of primary dysmenorrhea is elevated levels of uterine prostaglandins. Aetiological studies report that production of prostaglandins is controlled by the sex hormone progesterone, with prostaglandins and progesterone displaying an inverse relationship (i.e. increased progesterone levels reduce prostaglandin levels). Pro-inflammatory cytokines (interleukin-6 [IL-6] and tumor necrosis factor-alpha [TNF-α]) are also implicated in the pathogenesis of primary dysmenorrhea. High-intensity aerobic exercise is effective for decreasing pain quality and intensity in women with primary dysmenorrhea. However, why and how aerobic exercise is effective for treatment of primary dysmenorrhea remain unclear. Our preliminary non-randomized controlled pilot study to examine the effects of high-intensity aerobic exercise on progesterone, prostaglandin metabolite (13,14-dihydro-15-keto-prostaglandin F2 alpha (KDPGF Topics: Cytokines; Dinoprost; Dysmenorrhea; Exercise; Female; Hormones; Humans; Inflammation; Pain Management; Pilot Projects; Progesterone; Prostaglandins; Uterus | 2019 |