12-(1-pyrene)dodecanoic acid has been researched along with Zellweger Syndrome in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 3 (75.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Imanaka, T; Kawaguchi, K; Matsumoto, S; Matsuya, Y; Minato, D; Morita, M; Okazaki, A; Shimozawa, N; Tomita, K; Watanabe, S | 1 |
| Bogaki, A; Fujiki, Y; Okumoto, K; Orii, T; Osumi, T; Shimozawa, N; Suzuki, Y; Tateishi, K; Tsukamoto, T | 1 |
| Fujiki, Y; Kondo, N; Okano, I; Okumoto, K; Osumi, T; Shimozawa, N; Suzuki, Y; Tateishi, K; Tsukamoto, T | 1 |
| Calzolari, E; Cavazzoni, M; Griffoni, C; Spisni, E; Tomasi, V | 1 |
4 other study(ies) available for 12-(1-pyrene)dodecanoic acid and Zellweger Syndrome
| Article | Year |
|---|---|
A novel method for determining peroxisomal fatty acid β-oxidation.
Topics: Adrenoleukodystrophy; Carnitine Acyltransferases; Cells, Cultured; Fatty Acids; Fibroblasts; Humans; Lauric Acids; Lipid Metabolism, Inborn Errors; Mitochondria; Oxidation-Reduction; Peroxisomal Disorders; Peroxisomes; Pyrenes; Skin; Zellweger Syndrome | 2016 |
Isolation and characterization of peroxisome-deficient Chinese hamster ovary cell mutants representing human complementation group III.
Topics: Adenosine Triphosphatases; Animals; ATPases Associated with Diverse Cellular Activities; CHO Cells; Cricetinae; Drug Resistance; Genetic Complementation Test; Humans; Lauric Acids; Microbodies; Mutation; Peroxisomal Disorders; Zellweger Syndrome | 1997 |
Newly identified Chinese hamster ovary cell mutants defective in peroxisome biogenesis represent two novel complementation groups in mammals.
Topics: Acetyl-CoA C-Acyltransferase; Acyl-CoA Oxidase; Animals; Catalase; Cell Fusion; CHO Cells; Cricetinae; Cytosol; Digitonin; Fibroblasts; Genetic Complementation Test; Humans; Lauric Acids; Mammals; Membrane Proteins; Microbodies; Mutagenesis; Mutation; Oxidoreductases; Peroxisomal Biogenesis Factor 2; Ultraviolet Rays; Zellweger Syndrome | 1997 |
Evidence that photodynamic stress kills Zellweger fibroblasts by a nonapoptotic mechanism.
Topics: Apoptosis; Catalase; Cell Death; Cell Line; Cell Survival; Dose-Response Relationship, Drug; Ethidium; Fibroblasts; Flow Cytometry; Humans; Lauric Acids; Photochemotherapy; Poly(ADP-ribose) Polymerases; Proto-Oncogene Proteins c-bcl-2; Reactive Oxygen Species; Skin; Ultraviolet Rays; Zellweger Syndrome | 1998 |