1-methyltryptophan has been researched along with Kidney Diseases in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Čepcová, D; Deelman, LE; Kema, IP; Klimas, J; Sandovici, M; Šišková, K; Vavrincová-Yaghi, D; Vavrinec, P | 1 |
| Albejante, MC; Amaral, TA; Bach, EE; Brito, RBO; Camacho, CP; Dalboni, MA; Dellê, H; Malta, CS; Matheus, LHG; Matos, YST; Rodrigues, GGC; Santana, AC; Simão, GM | 1 |
2 other study(ies) available for 1-methyltryptophan and Kidney Diseases
| Article | Year |
|---|---|
The protective effect of 1-methyltryptophan isomers in renal ischemia-reperfusion injury is not exclusively dependent on indolamine 2,3-dioxygenase inhibition.
Topics: Animals; Coculture Techniques; Dendritic Cells; Disease Models, Animal; Enzyme Inhibitors; Epithelial-Mesenchymal Transition; Fibrosis; Humans; Indoleamine-Pyrrole 2,3,-Dioxygenase; Kidney; Kidney Diseases; Lectins, C-Type; Mice; NIH 3T3 Cells; NK Cell Lectin-Like Receptor Subfamily K; Reperfusion Injury; Signal Transduction; T-Lymphocytes; THP-1 Cells; Toll-Like Receptor 4; Transforming Growth Factor beta; Tryptophan | 2021 |
Indoleamine 2, 3-dioxygenase (IDO) increases during renal fibrogenesis and its inhibition potentiates TGF-β 1-induced epithelial to mesenchymal transition.
Topics: Animals; Dogs; Epithelial-Mesenchymal Transition; Fibrosis; Indoleamine-Pyrrole 2,3,-Dioxygenase; Kidney Diseases; Madin Darby Canine Kidney Cells; Male; Rats; Rats, Wistar; Transforming Growth Factor beta1; Tryptophan | 2017 |