1-methyltryptophan has been researched along with Cirrhosis in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 1 (33.33) | 2.80 |
Authors | Studies |
---|---|
Čepcová, D; Deelman, LE; Kema, IP; Klimas, J; Sandovici, M; Šišková, K; Vavrincová-Yaghi, D; Vavrinec, P | 1 |
Albejante, MC; Amaral, TA; Bach, EE; Brito, RBO; Camacho, CP; Dalboni, MA; Dellê, H; Malta, CS; Matheus, LHG; Matos, YST; Rodrigues, GGC; Santana, AC; Simão, GM | 1 |
Ahn, MS; Ahn, SG; Eom, YW; Kim, JY; Lee, JH; Lee, JW; Lee, SH; Oh, JE; Park, SW; Rhee, KJ; Son, JW; Yoo, BS; Yoon, J; Youn, YJ | 1 |
3 other study(ies) available for 1-methyltryptophan and Cirrhosis
Article | Year |
---|---|
The protective effect of 1-methyltryptophan isomers in renal ischemia-reperfusion injury is not exclusively dependent on indolamine 2,3-dioxygenase inhibition.
Topics: Animals; Coculture Techniques; Dendritic Cells; Disease Models, Animal; Enzyme Inhibitors; Epithelial-Mesenchymal Transition; Fibrosis; Humans; Indoleamine-Pyrrole 2,3,-Dioxygenase; Kidney; Kidney Diseases; Lectins, C-Type; Mice; NIH 3T3 Cells; NK Cell Lectin-Like Receptor Subfamily K; Reperfusion Injury; Signal Transduction; T-Lymphocytes; THP-1 Cells; Toll-Like Receptor 4; Transforming Growth Factor beta; Tryptophan | 2021 |
Indoleamine 2, 3-dioxygenase (IDO) increases during renal fibrogenesis and its inhibition potentiates TGF-β 1-induced epithelial to mesenchymal transition.
Topics: Animals; Dogs; Epithelial-Mesenchymal Transition; Fibrosis; Indoleamine-Pyrrole 2,3,-Dioxygenase; Kidney Diseases; Madin Darby Canine Kidney Cells; Male; Rats; Rats, Wistar; Transforming Growth Factor beta1; Tryptophan | 2017 |
Co-treatment with interferon-γ and 1-methyl tryptophan ameliorates cardiac fibrosis through cardiac myofibroblasts apoptosis.
Topics: Autophagy; Cell Cycle Checkpoints; Fibrosis; Gene Expression Regulation; Humans; Interferon-gamma; Muscle Proteins; Myocardial Infarction; Myocardium; Myofibroblasts; Signal Transduction; Tryptophan | 2019 |